Mechanisms of disease - Persistence of the Epstein-Barr virus and the origins of associated lymphomas

被引：734
作者：
Thorley-Lawson, DA
Gross, A
机构：
[1] Tufts Univ, Sch Med, Dept Pathol, Boston, MA 02111 USA
[2] Tufts Univ New England Med Ctr, Dept Rheumatol, Boston, MA USA
来源：
NEW ENGLAND JOURNAL OF MEDICINE | 2004年 / 350卷 / 13期
关键词：
D O I：
10.1056/NEJMra032015
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
[No abstract available]
引用
收藏
页码：1328 / 1337
页数：10
相关论文
共 78 条
[1]   MORPHOLOGY, IMMUNOPHENOTYPE, AND DISTRIBUTION OF LATENTLY AND/OR PRODUCTIVELY EPSTEIN-BARR VIRUS-INFECTED CELLS IN ACUTE INFECTIOUS-MONONUCLEOSIS - IMPLICATIONS FOR THE INTERINDIVIDUAL INFECTION ROUTE OF EPSTEIN-BARR-VIRUS [J].
ANAGNOSTOPOULOS, I ;
HUMMEL, M ;
KRESCHEL, C ;
STEIN, H .
BLOOD, 1995, 85 (03) :744-750
[2]   Tonsillar memory B cells, latently infected with Epstein-Barr virus, express the restricted pattern of latent genes previously found only in Epstein-Barr virus-associated tumors [J].
Babcock, GJ ;
Thorley-Lawson, DA .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2000, 97 (22) :12250-12255
[3]   The expression pattern of Epstein-Barr virus latent genes in vivo is dependent upon the differentiation stage of the infected B cell [J].
Babcock, GJ ;
Hochberg, D ;
Thorley-Lawson, DA .
IMMUNITY, 2000, 13 (04) :497-506
[4]   Epstein-Barr virus-infected resting memory B cells, not proliferating lymphoblasts, accumulate in the peripheral blood of immunosuppressed patients [J].
Babcock, GJ ;
Decker, LL ;
Freeman, RB ;
Thorley-Lawson, DA .
JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (04) :567-576
[5]   EBV persistence in memory B cells in vivo [J].
Babcock, GJ ;
Decker, LL ;
Volk, M ;
Thorley-Lawson, DA .
IMMUNITY, 1998, 9 (03) :395-404
[6]   Maintenance of serological memory by polyclonal activation of human memory B cells [J].
Bernasconi, NL ;
Traggiai, E ;
Lanzavecchia, A .
SCIENCE, 2002, 298 (5601) :2199-2202
[7]   Alternate replication in B cells and epithelial cells switches tropism of Epstein-Barr virus [J].
Borza, CM ;
Hutt-Fletcher, LM .
NATURE MEDICINE, 2002, 8 (06) :594-599
[8]   Epstein-Barr virus LMP2A drives B cell development and survival in the absence of normal B cell receptor signals [J].
Caldwell, RG ;
Wilson, JB ;
Anderson, SJ ;
Longnecker, R .
IMMUNITY, 1998, 9 (03) :405-411
[9]   Direct visualization of antigen-specific CD8+ T cells during the primary immune response to Epstein-Barr virus in vivo [J].
Callan, MFC ;
Tan, L ;
Annels, N ;
Ogg, GS ;
Wilson, JDK ;
O'Callaghan, CA ;
Steven, N ;
McMichael, AJ ;
Rickinson, AB .
JOURNAL OF EXPERIMENTAL MEDICINE, 1998, 187 (09) :1395-1402
[10]   B cell receptor signal strength determines B cell fate [J].
Casola, S ;
Otipoby, KL ;
Alimzhanov, M ;
Humme, S ;
Uyttersprot, N ;
Kutok, JL ;
Carroll, MC ;
Rajewsky, K .
NATURE IMMUNOLOGY, 2004, 5 (03) :317-327
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