ITAMs versus ITIMs: striking a balance during cell regulation

[21] A dual role for Src homology 2 domain-containing inositol-5-phosphatase (SHIP) in immunity:: Aberrant development and enhanced function of B lymphocytes in SHIP-/- mice [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 191 (05) : 781 - 794

[22] Isakov N, 1998, RECEPTOR CHANNEL, V5, P243

[23] Efficient cell activation requires an optimal dwell-time of interaction between the TCR and the pMHC complex [J]. NATURE IMMUNOLOGY, 2001, 2 (03) : 229 - 234

[24] Partially phosphorylated T cell receptor ζ molecules can inhibit T cell activation [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (11) : 1627 - 1636

[25] Fidelity of T cell activation through multistep T cell receptor ζ phosphorylation [J]. SCIENCE, 1998, 281 (5376) : 572 - 575

[26] Antigens varying in affinity for the B cell receptor induce differential B lymphocyte responses [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1998, 188 (08) : 1453 - 1464

[27] Interference with immunoglobulin (Ig)α immunoreceptor tyrosine-based activation motif (ITAM) phosphorylation modulates or blocks B cell development, depending on the availability of an Igβ cytoplasmic tail [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2001, 194 (04) : 455 - 469

[28] Identification of an immunoreceptor tyrosine-based activation motif of K1 transforming protein of Kaposi's sarcoma-associated herpesvirus [J]. MOLECULAR AND CELLULAR BIOLOGY, 1998, 18 (09) : 5219 - 5228

[29] SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival [J]. GENES & DEVELOPMENT, 1999, 13 (07) : 786 - 791

[30] IN SEARCH OF THE MISSING SELF - MHC MOLECULES AND NK CELL RECOGNITION [J]. IMMUNOLOGY TODAY, 1990, 11 (07): : 237 - 244

[21] A dual role for Src homology 2 domain-containing inositol-5-phosphatase (SHIP) in immunity:: Aberrant development and enhanced function of B lymphocytes in SHIP-/- mice [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2000, 191 (05) : 781 - 794

[22] Isakov N, 1998, RECEPTOR CHANNEL, V5, P243

[23] Efficient cell activation requires an optimal dwell-time of interaction between the TCR and the pMHC complex [J]. NATURE IMMUNOLOGY, 2001, 2 (03) : 229 - 234

[24] Partially phosphorylated T cell receptor ζ molecules can inhibit T cell activation [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (11) : 1627 - 1636

[25] Fidelity of T cell activation through multistep T cell receptor ζ phosphorylation [J]. SCIENCE, 1998, 281 (5376) : 572 - 575

[26] Antigens varying in affinity for the B cell receptor induce differential B lymphocyte responses [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1998, 188 (08) : 1453 - 1464

[27] Interference with immunoglobulin (Ig)α immunoreceptor tyrosine-based activation motif (ITAM) phosphorylation modulates or blocks B cell development, depending on the availability of an Igβ cytoplasmic tail [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 2001, 194 (04) : 455 - 469

[28] Identification of an immunoreceptor tyrosine-based activation motif of K1 transforming protein of Kaposi's sarcoma-associated herpesvirus [J]. MOLECULAR AND CELLULAR BIOLOGY, 1998, 18 (09) : 5219 - 5228

[29] SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival [J]. GENES & DEVELOPMENT, 1999, 13 (07) : 786 - 791

[30] IN SEARCH OF THE MISSING SELF - MHC MOLECULES AND NK CELL RECOGNITION [J]. IMMUNOLOGY TODAY, 1990, 11 (07): : 237 - 244