Human tissue kallikreins as promiscuous modulators of homeostatic skin barrier functions

被引:65
作者
Eissa, Azza [1 ,2 ]
Diamandis, Eleftherios P. [1 ,2 ,3 ,4 ]
机构
[1] Univ Toronto, Dept Lab Med & Pathobiol, Toronto, ON M5G 1L5, Canada
[2] Mt Sinai Hosp, Dept Pathol & Lab Med, Toronto, ON M5T 3L9, Canada
[3] Univ Hlth Network, Dept Clin Biochem, Toronto, ON M5G 1X5, Canada
[4] Toronto Med Labs, Toronto, ON M5G 1X5, Canada
关键词
activation cascade; corneodesmosomes; proteinase-activated receptors; serine protease inhibitors; skin diseases; stratum corneum;
D O I
10.1515/BC.2008.079
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Human tissue kallikreins (KLKs) are the largest family of secreted serine protease endopeptidases encoded by 15 genes clustered on chromosome 19q13.4. Multiple KLK enzymes are co-localized in the upper stratum granulosum and stratum corneum of human epidermis, and in associated appendages such as hair follicle epithelia and sweat glands. Until recently, kallikrein proteolytic activity in the skin was exclusively attributed to KLK5 and KLK7. However, wider cutaneous roles of kallikreins became evident in recent years as the proposal of KLK proteolytic activation cascades emerged. We postulate that these proteolytic enzymes may serve as promiscuous mediators of different skin barrier functions, since they are capable of proteolysing different substrates that govern skin desquamation, antimicrobial defense, and lipid permeability. Growing evidence now attests to potential kallikrein involvement in skin inflammation, pigmentation, and tumor suppression via their ability to target proteinase-activated receptor signaling pathways. Current knowledge on kallikrein roles in skin physiology and pathobiology is described in this review.
引用
收藏
页码:669 / 680
页数:12
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