Sustained Leukemia-Free State and Molecular Response to Sorafenib in a Patient With Chronic Myelomonocytic Leukemia in Transformation Driven by Homozygous FLT3-ITD Malignant Hematopoiesis

被引:2
作者
Kosmider, Olivier [1 ,2 ,3 ]
Chapuis, Nicolas [1 ,2 ,3 ]
Kaltenbach, Sophie [3 ,4 ]
Coriat, Romain [3 ]
Rouquette, Pascaline Boudou [3 ,5 ]
Willems, Lise [2 ,3 ,6 ]
Chesnais, Virginie [2 ,3 ]
Radford-Weiss, Isabelle [3 ,4 ]
Bardet, Valerie [1 ,2 ,3 ]
Mayeux, Patrick [2 ,3 ]
Tamburini, Jerome [2 ,3 ,6 ]
Fontenay, Michaela [1 ,2 ,3 ]
Bouscary, Didier [2 ,3 ,6 ]
机构
[1] Hop Cochin, AP HP, Serv Hematol Biol, F-75674 Paris, France
[2] CNRS, INSERM, Inst Cochin, Dept Immunohematol,UMR8104,U1016, Paris, France
[3] Univ Paris 05, Fac Med Sorbonne, Paris, France
[4] Hop Necker Enfants Malad, AP HP, Cytogenet Serv, Paris, France
[5] Hop Cochin, AP HP, Ctr Rech Inhibiteurs Angiogenese CERIA, F-75674 Paris, France
[6] Hop Cochin, AP HP, UF Hematol, F-75674 Paris, France
关键词
ASXL1; mutation; CMML; Targeted therapy; FLT3-ITD; Sorafenib; ACUTE MYELOID-LEUKEMIA; WILD-TYPE ALLELE; MYELODYSPLASTIC SYNDROMES; ASXL1; MUTATIONS; COMBINATION; CYTARABINE; MODEL;
D O I
10.1016/j.clml.2012.11.007
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
[No abstract available]
引用
收藏
页码:347 / 350
页数:4
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