Pathogenesis of endometriosis

被引：90

作者：

Oral, E ^{[1
]}

Arici, A ^{[1
]}

机构：

[1] YALE UNIV, SCH MED, DEPT OBSTET & GYNECOL, DIV REPROD ENDOCRINOL & INFERTIL, NEW HAVEN, CT 06520 USA

来源：

OBSTETRICS AND GYNECOLOGY CLINICS OF NORTH AMERICA | 1997年 / 24卷 / 02期

关键词：

D O I：

10.1016/S0889-8545(05)70301-6

中图分类号：

R71 [妇产科学];

学科分类号：

100211 ;

摘要：

This article reviews the pathogenesis of endometriosis, which involves retrograde dissemination of endometrium and/or development in situ by metaplasia, and there is evidence suggesting that genetic and hormonal factors may play a role. There is also substantial evidence that immunologic factors, namely peritoneal macrophages, T cells, natural killer cells, and soluble products secreted by these cells, are involved in the pathogenesis of endometriosis.

引用

页码：219 / +

页数：1

共 77 条

[1] The effect of endometriosis on implantation: Results from the Yale University in vitro fertilization and embryo transfer program [J].