Lentiviral Vif: Viral hijacker of the ubiquitin-proteasome system

被引：13

作者：

Ehrlich, ES ^{[1
]}

Yu, XF ^{[1
]}

机构：

[1] Johns Hopkins Bloomberg Sch Publ Hlth, Dept Mol Microbiol & Immunol, Baltimore, MD 21205 USA

来源：

INTERNATIONAL JOURNAL OF HEMATOLOGY | 2006年 / 83卷 / 03期

关键词：

Vif; APOBEC3; cytidine deaminase; HIV;

D O I：

10.1532/IJH97.06013

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Since the beginning of time, microorganisms have been devising ways to bypass detection and destruction by our immune system. Therefore, it is no surprise that along with the identification of the cellular antiviral protein APOBEC3G (A3G) has come the recognition of the viral solution to this assault. Here, we review the research that led up to the identification of A3G and the mechanism that the human immunodeficiency virus protein Vif developed to evade A3G's antiviral activities.

引用

页码：208 / 212

页数：5

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