Epigenetic regulation of hypoxia inducible factor in diseases and therapeutics

被引:36
作者
Minh Phuong Nguyen [1 ]
Lee, Sangkyu [2 ]
Lee, You Mie [2 ,3 ]
机构
[1] Kyungpook Natl Univ, Coll Pharm, Sch Life Sci & Biotechnol, Taegu 702701, South Korea
[2] Kyungpook Natl Univ, Coll Pharm, Pharmaceut Sci Res Inst, Taegu 702701, South Korea
[3] Kyungpook Natl Univ, Coll Pharm, Vasc Network Res Lab, Vasc Homeostasis Regulat Lab BRL, Taegu 702701, South Korea
关键词
Hypoxia-inducible factor; DNA methylation; Histone modification; Epigenetics; microRNAs; SQUAMOUS-CELL CARCINOMA; TUMOR-SUPPRESSOR GENE; HISTONE DEACETYLASE INHIBITOR; ENDOTHELIAL GROWTH-FACTOR; HIPPEL-LINDAU PROTEIN; HUMAN CANCER-CELLS; DNA METHYLATION; RNA INTERFERENCE; FACTOR; 1-ALPHA; TRANSCRIPTIONAL ACTIVITY;
D O I
10.1007/s12272-013-0058-x
中图分类号
R914 [药物化学];
学科分类号
100701 ;
摘要
Hypoxia-inducible factors (HIFs) are master regulators of angiogenesis and cellular adaptation in hypoxic microenvironments. Accumulating evidence indicates that HIFs also regulate cell survival, glucose metabolism, microenvironmental remodeling, cancer metastasis, and tumor progression, and thus, HIFs are viewed as therapeutic targets in many diseases. Epigenetic changes are involved in the switching 'on' and 'off' of many genes, and it has been suggested that the DNA hypermethylation of specific gene promoters, histone modifications (acetylation, phosphorylation, and methylation) and small interfering or micro RNAs be regarded epigenetic gene targets for the regulation of disease-associated cellular changes. Furthermore, the hypoxic microenvironment is one of the most important cellular stress stimuli in terms of the regulation of cellular epigenetic status via histone modification. Therefore, drug development and therapeutic approaches to ischemic diseases or cancer for targeting HIFs by modulation of epigenetic status become an attractive area. Here, the authors provide a review of the literature regarding the targeting of HIF, a key modulator of hypoxic-cell response under various disease conditions, by modulating histone or DNA using endogenous small RNAs or exogenous chemicals.
引用
收藏
页码:252 / 263
页数:12
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