Hyperglycemia triggers epigenetic machinery to enhance MyD88 expression in macrophages from type 1 diabetes mice

被引:0
|
作者
Filgueiras, Luciano [1 ,2 ]
Sisti, Flavia [1 ,3 ]
Ferreira, Ana [1 ,3 ]
Wang, Soujuan [1 ]
Wang, Zhuo [1 ]
Jancar, Sonia [2 ]
Serezani, C. Henrique [1 ]
机构
[1] Indiana Univ, Sch Med, Indianapolis, IN USA
[2] Univ Sao Paulo, Inst Biomed Sci, Sao Paulo, Brazil
[3] Univ Sao Paulo, Sch Med, BR-14049 Ribeirao Preto, Brazil
来源
JOURNAL OF IMMUNOLOGY | 2013年 / 190卷
关键词
D O I
暂无
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
P1350
引用
收藏
页数:1
相关论文
共 45 条
  • [1] Epigenetic regulation of the overexpressed STAT-1/Myd88 in macrophages from type 1 diabetic mice
    Ribeiro Filgueiras, L.
    Brandt, S.
    Raquel Oliveira Ramalho, T.
    Jancar, S.
    Henrique Serezani, C.
    EUROPEAN JOURNAL OF IMMUNOLOGY, 2016, 46 : 855 - 856
  • [2] MyD88 mediated environmental effect on type 1 diabetes development in NOD mice
    Chervonsky, A.
    Hu, C. Y.
    Du, W.
    Lu, B.
    Zhang, L.
    Wong, S. F.
    Wen, L.
    ACTA DIABETOLOGICA, 2007, 44 : S54 - S54
  • [3] Imbalance between HDAC and HAT activities drives aberrant STAT1/MyD88 expression in macrophages from type 1 diabetic mice
    Filgueiras, Luciano Ribeiro
    Brandt, Stephanie L.
    de Oliveira Ramalho, Theresa Raquel
    Jancar, Sonia
    Henrique Serezani, C.
    JOURNAL OF DIABETES AND ITS COMPLICATIONS, 2017, 31 (02) : 334 - 339
  • [4] Beyond knockout A novel homodimerization-targeting MyD88 inhibitor prevents and cures type 1 diabetes in NOD mice
    Zhang, Xue
    Xing, Shuai
    Li, Mingqiang
    Zhang, Limin
    Xie, Lin
    He, Wentao
    Liu, Jianhua
    Chang, Sheng
    Jiang, Fengchao
    Zhou, Ping
    METABOLISM-CLINICAL AND EXPERIMENTAL, 2016, 65 (09): : 1267 - 1277
  • [5] IN TYPE 1 DIABETES MELLITUS IMPAIRED VASCULAR FUNCTION RELATES TO THE EXPRESSION OF MYD88 IN LYMPHOMONONUCLEAR CELLS BUT NOT TO DIETARY ADVANCED GLYCATION END-PRODUCTS
    Toyoshima, M. T. K.
    Santos-Bezerra, D. P.
    Machado-Lima, A.
    Goes, M. F. S.
    Caramelli, B.
    Correa-Giannella, M. L. C.
    Passarelli, M.
    ATHEROSCLEROSIS, 2018, 275 : E63 - E63
  • [6] In vivo PPAR-gamma activation inhibits MyD88 expression to protect mice from polymicrobial sepsis.
    Ferreira, Ana
    Sisti, Flavia
    Filgueiras, Luciano
    Wang, Sue
    Wang, Julia
    Cunha, Fernando
    Alves-Filho, Jose
    Serezani, C. Henrique
    JOURNAL OF IMMUNOLOGY, 2013, 190
  • [7] A MyD88–JAK1–STAT1 complex directly induces SOCS-1 expression in macrophages infected with Group A Streptococcus
    Jinghua Wu
    Cuiqing Ma
    Haixin Wang
    Shuhui Wu
    Gao Xue
    Xinli Shi
    Zhang Song
    Lin Wei
    Cellular & Molecular Immunology, 2015, 12 : 373 - 383
  • [8] Mice deficient in MyD88 display profound defects in acute resistance and type 1 cytokine responses to Mycobacterium avium infection
    Scanga, CA
    Feng, CG
    Collazo-Custodio, CM
    Cheever, AW
    Sher, A
    FASEB JOURNAL, 2003, 17 (07): : C288 - C288
  • [9] RETRACTED ARTICLE: SP1-induced lncRNA AGAP2-AS1 expression promotes chemoresistance of breast cancer by epigenetic regulation of MyD88
    Huaying Dong
    Wei Wang
    Shaowei Mo
    Ru Chen
    Kejian Zou
    Jing Han
    Fan Zhang
    Jianguo Hu
    Journal of Experimental & Clinical Cancer Research, 37
  • [10] Retraction Note: SP1-induced lncRNA AGAP2-AS1 expression promotes chemoresistance of breast cancer by epigenetic regulation of MyD88
    Huaying Dong
    Wei Wang
    Shaowei Mo
    Ru Chen
    Kejian Zou
    Jing Han
    Fan Zhang
    Jianguo Hu
    Journal of Experimental & Clinical Cancer Research, 42