The Histone Modification Code in the Pathogenesis of Autoimmune Diseases

被引:61
作者
Araki, Yasuto [1 ,2 ]
Mimura, Toshihide [1 ,2 ]
机构
[1] Saitama Med Univ, Dept Rheumatol & Appl Immunol, Fac Med, Saitama, Japan
[2] Saitama Med Univ, Res Ctr Genom Med, Project Res Div, Saitama, Japan
关键词
SYSTEMIC-LUPUS-ERYTHEMATOSUS; BLOOD MONONUCLEAR-CELLS; GENOME-WIDE ANALYSIS; RHEUMATOID-ARTHRITIS; T-CELLS; POSTTRANSLATIONAL MODIFICATIONS; ACETYLTRANSFERASE COMPLEXES; INTERLEUKIN-6; PRODUCTION; EPIGENETIC REPRESSION; ARGININE METHYLATION;
D O I
10.1155/2017/2608605
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
Autoimmune diseases are chronic inflammatory disorders caused by a loss of self-tolerance, which is characterized by the appearance of autoantibodies and/or autoreactive lymphocytes and the impaired suppressive function of regulatory T cells. The pathogenesis of autoimmune diseases is extremely complex and remains largely unknown. Recent advances indicate that environmental factors trigger autoimmune diseases in genetically predisposed individuals. In addition, accumulating results have indicated a potential role of epigenetic mechanisms, such as histone modifications, in the development of autoimmune diseases. Histone modifications regulate the chromatin states and gene transcription without any change in the DNA sequence, possibly resulting in phenotype alteration in several different cell types. In this paper, we discuss the significant roles of histone modifications involved in the pathogenesis of autoimmune diseases, including rheumatoid arthritis, systemic lupus erythematosus, systemic sclerosis, primary biliary cirrhosis, and type 1 diabetes.
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页数:12
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