The Structure, Function, and Physiology of the Fetal and Adult Acetylcholine Receptor in Muscle

被引:50
作者
Cetin, Hakan [1 ,2 ]
Beeson, David [2 ]
Vincent, Angela [2 ]
Webster, Richard [2 ]
机构
[1] Med Univ Vienna, Dept Neurol, Vienna, Austria
[2] Univ Oxford, Nuffield Dept Clin Neurosci, Oxford, England
来源
FRONTIERS IN MOLECULAR NEUROSCIENCE | 2020年 / 13卷
基金
英国医学研究理事会; 奥地利科学基金会;
关键词
fetal acetylcholine receptor; adult acetylcholine receptor; ion channel; myasthenia; neuromuscular junction; muscle development; ARTHROGRYPOSIS MULTIPLEX CONGENITA; EPSILON-SUBUNIT GENE; ACHR DEFICIENCY SYNDROME; MESSENGER-RNA LEVELS; CHANNEL OPEN TIME; PROTEIN-KINASE-C; MYASTHENIA-GRAVIS; SKELETAL-MUSCLE; END-PLATE; NEUROMUSCULAR-JUNCTION;
D O I
10.3389/fnmol.2020.581097
中图分类号
Q189 [神经科学];
学科分类号
071006 ;
摘要
The neuromuscular junction (NMJ) is a highly developed synapse linking motor neuron activity with muscle contraction. A complex of molecular cascades together with the specialized NMJ architecture ensures that each action potential arriving at the motor nerve terminal is translated into an action potential in the muscle fiber. The muscle-type nicotinic acetylcholine receptor (AChR) is a key molecular component located at the postsynaptic muscle membrane responsible for the generation of the endplate potential (EPP), which usually exceeds the threshold potential necessary to activate voltage-gated sodium channels and triggers a muscle action potential. Two AChR isoforms are found in mammalian muscle. The fetal isoform is present in prenatal stages and is involved in the development of the neuromuscular system whereas the adult isoform prevails thereafter, except after denervation when the fetal form is re-expressed throughout the muscle. This review will summarize the structural and functional differences between the two isoforms and outline congenital and autoimmune myasthenic syndromes that involve the isoform specific AChR subunits.
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页数:14
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