共 53 条
Identification of novel markers of alternative activation and potential endogenous PPARγ ligand production mechanisms in human IL-4 stimulated differentiating macrophages
被引:38
作者:

Czimmerer, Zsolt
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Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary

Varga, Tamas
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Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary

Poliska, Szilard
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Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary
Univ Debrecen, Med & Hlth Sci Ctr, Debrecen Clin Genom Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary

Nemet, Istvan
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Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary

Szanto, Attila
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机构:
Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary

Nagy, Laszlo
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h-index: 0
机构:
Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary
Univ Debrecen, MTA DE Lendulet Immungen Res Grp, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary
机构:
[1] Univ Debrecen, Dept Biochem & Mol Biol, Res Ctr Mol Med, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary
[2] Univ Debrecen, MTA DE Lendulet Immungen Res Grp, Med & Hlth Sci Ctr, H-4012 Debrecen, Hungary
[3] Univ Debrecen, Med & Hlth Sci Ctr, Debrecen Clin Genom Ctr, H-4012 Debrecen, Hungary
基金:
匈牙利科学研究基金会;
关键词:
Alternative macrophage activation markers;
ENPP2;
MAOA;
PPAR gamma ligand;
Serotonin;
TUMOR-ASSOCIATED MACROPHAGES;
FACTOR PATHWAY INHIBITOR-2;
T-CELLS;
GENE-EXPRESSION;
DENDRITIC CELLS;
HUMAN MONOCYTES;
ADIPOSE-TISSUE;
RECEPTOR;
CANCER;
INFLAMMATION;
D O I:
10.1016/j.imbio.2012.08.270
中图分类号:
R392 [医学免疫学];
Q939.91 [免疫学];
学科分类号:
100102 ;
摘要:
We analyzed global gene expression profiles of IL-4 induced alternatively activated as well as IFN-gamma+TNF alpha stimulated classically activated human monocyte derived macrophages and identified novel IL-4 regulated alternative activation marker genes including MS4A4A, SLA, CD180, and ENPP2. Transcription factor prediction analysis of IL-4 regulated genes suggested that the regulated genes are involved in a complex regulation of lipid metabolism, defense against cell metabolism derived reactive oxygen species, and basal expression of inflammation linked genes. Both an in silico transcription activation prediction as well as experimental data suggested the presence of alternative macrophage activation specific endogenous PPAR gamma ligand producing mechanisms. We found the induction of three enzymes whose activity can potentially generate endogenous PPAR gamma ligands in an IL-4 dependent manner. These are MAOA, ENPP2, and ALOX15 producing 5-methoxy-indole acetate, lysophosphatidic acid (LPA) and 13-hydroxyoctadienoic acid (13-HODE), and/or 15-hydroxyeicosatetraenoic acid (15-HETE), respectively. Our data suggest that global gene expression profiling, combined with computational transcription activity prediction, can lead to identification of transcriptional networks that underpin cellular subtype specification. (C) 2012 Elsevier GmbH. All rights reserved.
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页码:1301 / 1314
页数:14
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Quinnies, Kayla M.
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Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA

Crihfield, Anastasia
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Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA

Lynch, Kevin R.
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机构:
Univ Virginia, Dept Pharmacol, Charlottesville, VA 22908 USA Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA

Kipnis, Jonathan
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机构:
Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA
Univ Virginia, Grad Program Neurosci, Charlottesville, VA 22908 USA
Univ Virginia, Grad Program Immunol, Charlottesville, VA 22908 USA Univ Virginia, Dept Neurosci, Charlottesville, VA 22908 USA
[10]
Negative regulation of Toll-like receptor 4 signaling by the Toll-like receptor homolog RP105
[J].
Divanovic, S
;
Trompette, A
;
Atabani, SF
;
Madan, R
;
Golenbock, DT
;
Visintin, A
;
Finberg, RW
;
Tarakhovsky, A
;
Vogel, SN
;
Belkaid, Y
;
Kurt-Jones, EA
;
Karp, CL
.
NATURE IMMUNOLOGY,
2005, 6 (06)
:571-578

Divanovic, S
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Trompette, A
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h-index: 0
机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Atabani, SF
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h-index: 0
机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Madan, R
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h-index: 0
机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Golenbock, DT
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h-index: 0
机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Visintin, A
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Finberg, RW
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Tarakhovsky, A
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Vogel, SN
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Belkaid, Y
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Kurt-Jones, EA
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机构: Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA

Karp, CL
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机构:
Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA Cincinnati Childrens Hosp, Med Ctr, Div Mol Immunol, Cincinnati, OH 45229 USA