EAST/SeSAME syndrome mutations partially disrupt trafficking of homomeric Kir4.1 channels and produce non-functional heteromeric Kir2.3/Kir4.1 channels

被引：0

作者：

Ortega, Bernardo ^{[1
]}

Li, Dimin ^{[1
]}

Kim, Bo-Young ^{[1
]}

Wade, James B. ^{[1
]}

Welling, Paul A. ^{[1
]}

机构：

[1] Univ Maryland, Sch Med, Baltimore, MD 21201 USA

来源：

FASEB JOURNAL | 2012年 / 26卷

关键词：

D O I：

暂无

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页数：1

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