EAST/SeSAME syndrome mutations partially disrupt trafficking of homomeric Kir4.1 channels and produce non-functional heteromeric Kir2.3/Kir4.1 channels

被引：0
作者：
Ortega, Bernardo [1 ]
Li, Dimin [1 ]
Kim, Bo-Young [1 ]
Wade, James B. [1 ]
Welling, Paul A. [1 ]
机构：
[1] Univ Maryland, Sch Med, Baltimore, MD 21201 USA
来源：
FASEB JOURNAL | 2012年 / 26卷
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D O I：
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中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
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页数：1
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