EAST/SeSAME syndrome mutations partially disrupt trafficking of homomeric Kir4.1 channels and produce non-functional heteromeric Kir2.3/Kir4.1 channels

被引:0
作者
Ortega, Bernardo [1 ]
Li, Dimin [1 ]
Kim, Bo-Young [1 ]
Wade, James B. [1 ]
Welling, Paul A. [1 ]
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[1] Univ Maryland, Sch Med, Baltimore, MD 21201 USA
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Q5 [生物化学]; Q7 [分子生物学];
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071010 ; 081704 ;
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页数:1
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