Light-Controlled Toxicity of Engineered Amyloid β-Peptides

[11] Fortin DL, 2008, NAT METHODS, V5, P331, DOI [10.1038/NMETH.1187, 10.1038/nmeth.1187]

[12] Role of Amyloid-β Glycine 33 in Oligomerization, Toxicity, and Neuronal Plasticity [J]. JOURNAL OF NEUROSCIENCE, 2009, 29 (23) : 7582 - 7590

[13] Hoppmann C., 2011, ANGEW CHEM, V123, P7841

[14] Photocontrol of Contracting Muscle Fibers [J]. ANGEWANDTE CHEMIE-INTERNATIONAL EDITION, 2011, 50 (33) : 7699 - 7702

[15] Aβ42 Neurotoxicity Is Mediated by Ongoing Nucleated Polymerization Process Rather than by Discrete Aβ42 Species [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2011, 286 (10) : 8585 - 8596

[16] Study of the conformational transition of Aβ(1-42) using D-amino acid replacement analogues [J]. BIOCHEMISTRY, 2001, 40 (18) : 5457 - 5463

[17] THE PRECURSOR OF ALZHEIMERS-DISEASE AMYLOID-A4 PROTEIN RESEMBLES A CELL-SURFACE RECEPTOR [J]. NATURE, 1987, 325 (6106) : 733 - 736

[18] Amyloid β protein forms ion channels:: implications for Alzheimer's disease pathophysiology [J]. FASEB JOURNAL, 2001, 15 (13) : 2433 - 2444

[19] Lipids revert inert Aβ amyloid fibrils to neurotoxic protofibrils that affect learning in mice [J]. EMBO JOURNAL, 2008, 27 (01) : 224 - 233

[20] Switch peptides in statu nascendi:: Induction of conformational transitions relevant to degenerative diseases [J]. ANGEWANDTE CHEMIE-INTERNATIONAL EDITION, 2004, 43 (32) : 4172 - 4178

[11] Fortin DL, 2008, NAT METHODS, V5, P331, DOI [10.1038/NMETH.1187, 10.1038/nmeth.1187]

[12] Role of Amyloid-β Glycine 33 in Oligomerization, Toxicity, and Neuronal Plasticity [J]. JOURNAL OF NEUROSCIENCE, 2009, 29 (23) : 7582 - 7590

[13] Hoppmann C., 2011, ANGEW CHEM, V123, P7841

[14] Photocontrol of Contracting Muscle Fibers [J]. ANGEWANDTE CHEMIE-INTERNATIONAL EDITION, 2011, 50 (33) : 7699 - 7702

[15] Aβ42 Neurotoxicity Is Mediated by Ongoing Nucleated Polymerization Process Rather than by Discrete Aβ42 Species [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2011, 286 (10) : 8585 - 8596

[16] Study of the conformational transition of Aβ(1-42) using D-amino acid replacement analogues [J]. BIOCHEMISTRY, 2001, 40 (18) : 5457 - 5463

[17] THE PRECURSOR OF ALZHEIMERS-DISEASE AMYLOID-A4 PROTEIN RESEMBLES A CELL-SURFACE RECEPTOR [J]. NATURE, 1987, 325 (6106) : 733 - 736

[18] Amyloid β protein forms ion channels:: implications for Alzheimer's disease pathophysiology [J]. FASEB JOURNAL, 2001, 15 (13) : 2433 - 2444

[19] Lipids revert inert Aβ amyloid fibrils to neurotoxic protofibrils that affect learning in mice [J]. EMBO JOURNAL, 2008, 27 (01) : 224 - 233

[20] Switch peptides in statu nascendi:: Induction of conformational transitions relevant to degenerative diseases [J]. ANGEWANDTE CHEMIE-INTERNATIONAL EDITION, 2004, 43 (32) : 4172 - 4178