Systemic lupus erythematosus and dysregulated apoptosis - what is the evidence?

被引：18

作者：

McHugh, NJ ^{[1
]}

机构：

[1] Royal Natl Hosp Rheumat Dis, Bath BA1 1RL, Avon, England

来源：

RHEUMATOLOGY | 2002年 / 41卷 / 03期

关键词：

D O I：

10.1093/rheumatology/41.3.242

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：242 / 245

页数：4

共 34 条

[1] Serum amyloid P component controls chromatin degradation and prevents antinuclear autoimmunity [J].

Bickerstaff, MCM ;

Botto, M ;

Hutchinson, WL ;

Herbert, J ;

Tennent, GA ;

Bybee, A ;

Mitchell, DA ;

Cook, HT ;

Butler, PJG ;

Walport, MJ ;

Pepys, MB .

NATURE MEDICINE, 1999, 5 (06) :694-697

[2] Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies [J].

Botto, M ;

Dell'Agnola, C ;

Bygrave, AE ;

Thompson, EM ;

Cook, HT ;

Petry, F ;

Loos, M ;

Pandolfi, PP ;

Walport, MJ .

NATURE GENETICS, 1998, 19 (01) :56-59

[3] Cleavage by granzyme B is strongly predictive of autoantigen status: Implications for initiation of autoimmunity [J].

Casciola-Rosen, L ;

Andrade, F ;

Ulanet, D ;

Wong, WB ;

Rosen, A .

JOURNAL OF EXPERIMENTAL MEDICINE, 1999, 190 (06) :815-825

[4] AUTOANTIGENS TARGETED IN SYSTEMIC LUPUS-ERYTHEMATOSUS ARE CLUSTERED IN 2 POPULATIONS OF SURFACE-STRUCTURES ON APOPTOTIC KERATINOCYTES [J].

CASCIOLAROSEN, LA ;

ANHALT, G ;

ROSEN, A .

JOURNAL OF EXPERIMENTAL MEDICINE, 1994, 179 (04) :1317-1330

[5] PROTECTION FROM FAS-MEDIATED APOPTOSIS BY A SOLUBLE FORM OF THE FAS MOLECULE [J].

CHENG, JH ;

ZHOU, T ;

LIU, CD ;

SHAPIRO, JP ;

BRAUER, MJ ;

KIEFER, MC ;

BARR, PJ ;

MOUNTZ, JD .

SCIENCE, 1994, 263 (5154) :1759-1762

[6]

Davies EJ, 1997, J RHEUMATOL, V24, P485

[7]

EMLEN W, 1994, J IMMUNOL, V152, P3685

[8] The biochemistry of apoptosis [J].

Hengartner, MO .

NATURE, 2000, 407 (6805) :770-776

[9]

Hieronymus T, 2000, ARTHRITIS RHEUM, V43, P1994, DOI 10.1002/1529-0131(200009)43:9<1994::AID-ANR10>3.0.CO

[10]

2-7

← 1 2 3 4 →