Targeting a single mismatched minor histocompatibility antigen with tumor-restricted expression eradicates human solid tumors

被引:25
作者
Hambach, Lothar [1 ]
Vermeij, Marcel [2 ]
Buser, Andreas [1 ]
Aghai, Zohara [1 ]
van der Kwast, Theodorus [2 ]
Goulmy, Els [1 ]
机构
[1] Leiden Univ, Med Ctr, Dept Immunohematol & Blood Transfus, NL-2300 RC Leiden, Netherlands
[2] Erasmus MC, Dept Pathol, Rotterdam, Netherlands
关键词
D O I
10.1182/blood-2007-11-125492
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Regressions of metastatic solid tumors after allogeneic human leukocyte antigen (HLA)-matched stem cell transplantation (SCT) are often associated with detrimental graft-versus-host disease (GVHD). The graft-versus-host reaction of the HLA-matched donor is directed mainly against the multiple mismatched minor histocompatibility antigens (mHags) of the patient. mHags are strong HLA-restricted alloantigens with differential tissue distribution. Ubiquitously expressed mHags are the prime in situ targets of GVHD. The mHag HA-1 is hematopoiesis restricted, but displays additionally an aberrant expression on solid tumors. Thus, HA-1 might be an excellent target to boost the anti-solid tumor effect of allogeneic SCT without inducing severe GVHD. Here, we show that cytotoxic T lymphocytes (CTLs) solely targeting the human mHag HA-1 are capable of eradicating 3-dimensional human solid tumors in a highly mHag-specific manner in vitro, accompanied by interferon-gamma release. In vivo, HA-1-specific CTLs distribute systemically and prevent human breast cancer metastases in immunodeficient mice. Moreover, HA-1-specific CTLs infiltrate and inhibit the progression of fully established metastases. Our study provides the first proof for the efficacy of a clinically applicable concept to exploit single mismatched mHags with hematopoiesis- and solid tumor-restricted expression for boosting the anti-solid tumor effect of allogeneic SCT.
引用
收藏
页码:1844 / 1852
页数:9
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