COVID-19: a cause of recurrent Graves' hyperthyroidism?

被引:59
作者
Jimenez-Blanco, S. [1 ]
Pla-Peris, B. [2 ]
Marazuela, M. [1 ]
机构
[1] Univ Hosp La Princesa, Dept Endocrinol & Nutr, Madrid, Spain
[2] Hosp Arnau Vilanova, Dept Endocrinol & Nutr, Valencia, Spain
关键词
D O I
10.1007/s40618-020-01440-0
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:387 / 388
页数:2
相关论文
共 5 条
[1]  
Barutta Federica, 2014, ENDOCRINE, V48, P730, DOI [10.1007/s12020-014-0437-1, DOI 10.1007/S12020-014-0437-1]
[2]   The pathogenesis of thyroid autoimmune diseases: New T lymphocytes - Cytokines circuits beyond the Th1-Th2 paradigm [J].
Li, Qian ;
Wang, Bin ;
Mu, Kaida ;
Zhang, Jin-An .
JOURNAL OF CELLULAR PHYSIOLOGY, 2019, 234 (03) :2204-2216
[3]   SARS-COV-2 as a trigger for autoimmune disease: report of two cases of Graves' disease after COVID-19 [J].
Mateu-Salat, M. ;
Urgell, E. ;
Chico, A. .
JOURNAL OF ENDOCRINOLOGICAL INVESTIGATION, 2020, 43 (10) :1527-1528
[4]   The environment and autoimmune thyroid diseases [J].
Prummel, MF ;
Strieder, T ;
Wiersinga, WM .
EUROPEAN JOURNAL OF ENDOCRINOLOGY, 2004, 150 (05) :605-618
[5]   Increased serum concentrations of interleukin-6 (IL-6) and soluble IL-6 receptor in patients with Graves' disease [J].
Salvi, M ;
Girasole, G ;
Pedrazzoni, M ;
Passeri, M ;
Giuliani, N ;
Minelli, R ;
Braverman, LE ;
Roti, E .
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1996, 81 (08) :2976-2979