Mitochondria and oxidative damage in amyotrophic lateral sclerosis

[1] BEAL MF, 1997, ANN NEUROL, V42, P646

[2] ALS, SOD AND PEROXYNITRITE [J]. NATURE, 1993, 364 (6438) : 584 - 584

[3] The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS, 1996, 1275 (1-2): : 5 - 9

[4] Complexes between hexokinase, mitochondrial porin and adenylate translocator in brain: Regulation of hexokinase, oxidative phosphorylation and permeability transition pore [J]. BIOCHEMICAL SOCIETY TRANSACTIONS, 1997, 25 (01) : 151 - 157

[5] Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore.: Implication for regulation of permeability transition by the kinases [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES, 1998, 1368 (01): : 7 - 18

[6] Increased oxidative damage to DNA in ALS patients [J]. FREE RADICAL BIOLOGY AND MEDICINE, 2000, 29 (07) : 652 - 658

[7] SUPEROXIDE-DISMUTASE-1 WITH MUTATIONS LINKED TO FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS POSSESSES SIGNIFICANT ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (17) : 8292 - 8296

[8] Borthwick GM, 1999, ANN NEUROL, V46, P787, DOI 10.1002/1531-8249(199911)46:5<787::AID-ANA17>3.0.CO

[9] 2-8

[10] SUPEROXIDE-DISMUTASE ACTIVITY, OXIDATIVE DAMAGE, AND MITOCHONDRIAL ENERGY-METABOLISM IN FAMILIAL AND SPORADIC AMYOTROPHIC-LATERAL-SCLEROSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (06) : 2322 - 2325

[1] BEAL MF, 1997, ANN NEUROL, V42, P646

[2] ALS, SOD AND PEROXYNITRITE [J]. NATURE, 1993, 364 (6438) : 584 - 584

[3] The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS, 1996, 1275 (1-2): : 5 - 9

[4] Complexes between hexokinase, mitochondrial porin and adenylate translocator in brain: Regulation of hexokinase, oxidative phosphorylation and permeability transition pore [J]. BIOCHEMICAL SOCIETY TRANSACTIONS, 1997, 25 (01) : 151 - 157

[5] Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore.: Implication for regulation of permeability transition by the kinases [J]. BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES, 1998, 1368 (01): : 7 - 18

[6] Increased oxidative damage to DNA in ALS patients [J]. FREE RADICAL BIOLOGY AND MEDICINE, 2000, 29 (07) : 652 - 658

[7] SUPEROXIDE-DISMUTASE-1 WITH MUTATIONS LINKED TO FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS POSSESSES SIGNIFICANT ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (17) : 8292 - 8296

[8] Borthwick GM, 1999, ANN NEUROL, V46, P787, DOI 10.1002/1531-8249(199911)46:5<787::AID-ANA17>3.0.CO

[9] 2-8

[10] SUPEROXIDE-DISMUTASE ACTIVITY, OXIDATIVE DAMAGE, AND MITOCHONDRIAL ENERGY-METABOLISM IN FAMILIAL AND SPORADIC AMYOTROPHIC-LATERAL-SCLEROSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (06) : 2322 - 2325