MicroRNA hsa-miR-665 prevents cardiomyocyte hypertrophy and preserves normal cardiac function after pressure overload

被引：0
作者：
Braga, L. [1 ]
Dal Ferro, M. [1 ,2 ]
Mano, M. [1 ]
Eulalio, A. [1 ]
Moimas, S. [1 ]
Zentilin, L. [1 ]
Zacchigna, S. [1 ]
Giacca, M. [1 ]
机构：
[1] ICGEB, Mol Med Lab, Trieste, Italy
[2] Azienda Sanit Univ Integrata Trieste, Dept Cardiol, Trieste, Italy
来源：
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY | 2017年 / 109卷
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
2
引用
收藏
页码：1 / 2
页数：2
相关论文
共 30 条
  • [21] Physiological Overexpression of PGC-1alpha Preserves Mitochondrial Function and Reduces Fibrosis but Does Not Prevent Cardiac Dysfunction Following Pressure Overload Hypertrophy
    Pereira, Renata O.
    Crum, Ashley
    Wende, Adam R.
    Olsen, Curtis D.
    Abel, E. Dale
    CIRCULATION, 2011, 124 (21)
  • [22] Orai1 Channel Inhibition Preserves Left Ventricular Systolic Function and Normal Ca2+ Handling After Pressure Overload
    Bartoli, Fiona
    Bailey, Marc A.
    Rode, Baptiste
    Mateo, Philippe
    Antigny, Fabrice
    Bedouet, Kaveen
    Gerbaud, Pascale
    Gosain, Rajendra
    Plante, Jeffrey
    Norman, Katherine
    Gomez, Susana
    Lefebvre, Florence
    Rucker-Martin, Catherine
    Ainscough, Justin F. X.
    Kearney, Mark T.
    Bruns, Alexander-Francisco
    Shi, Jian
    Appleby, Hollie L.
    Young, Richard S.
    Shawer, Heba M.
    Debant, Marjolaine
    Gomez, Ana-Maria
    Beech, David J.
    Foster, Richard
    Benitah, Jean-Pierre
    Sabourin, Jessica
    CIRCULATION, 2020, 141 (03) : 199 - 216
  • [23] Cardiomyocyte specific overexpression of a 37 amino acid domain of regulator of G protein signalling 2 inhibits cardiac hypertrophy and improves function in response to pressure overload in mice
    Lee, Katherine N.
    Lu, Xiangru
    Nguyen, Chau
    Feng, Qingping
    Chidiac, Peter
    JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 2017, 108 : 194 - 202
  • [24] Divergent effects of a Nox2-containing NADPH oxidase on cardiac contractile function and hypertrophy after imposition of chronic pressure overload
    Grieve, DJ
    Siva, A
    Byrne, JA
    Cave, AC
    Shah, AM
    CIRCULATION, 2004, 110 (17) : 133 - 134
  • [25] Short-Term Increase in Glut1-Mediated Myocardial Glucose Uptake Preserves Mitochondrial Function but Does Not Prevent Cardiac Dysfunction Following Pressure Overload Hypertrophy
    Pereira, Renata O.
    Wende, Adam R.
    Soto, Jamie
    Abel, E. Dale
    DIABETES, 2011, 60 : A409 - A409
  • [26] Mineralocorticoid receptor blockade arrests pressure overload-induced left ventricular hypertrophy and preserves cardiac function in angiotensin II type 1a receptor knockout mice
    Meguro, Tomomi
    Yoshikawa, Tsutomu
    Katada, Jun
    Saito, Hitomi
    Anzai, Toshihisa
    Nagatomo, Yuji
    Ogawa, Satoshi
    CIRCULATION, 2006, 114 (18) : 150 - 150
  • [27] Letter by Mehmood Regarding Article, "Orai1 Channel Inhibition Preserves Left Ventricular Systolic Function and Normal Ca2+Handling After Pressure Overload"
    Mehmood, Muddassir
    CIRCULATION, 2020, 141 (22) : E837 - E838
  • [28] Cardiomyocyte-specific miR-100 overexpression preserves heart function under pressure overload in mice and diminishes fatty acid uptake as well as ROS production by direct suppression of Nox4 and CD36
    Smolka, Christian
    Schloesser, Delia
    Koentges, Christoph
    Tarkhnishvili, Aleksandre
    Gorka, Oliver
    Pfeifer, Dietmar
    Bemtgen, Xavier
    Asmussen, Alexander
    Gross, Olaf
    Diehl, Philipp
    Moser, Martin
    Bode, Christoph
    Bugger, Heiko
    Grundmann, Sebastian
    Pankratz, Franziska
    FASEB JOURNAL, 2021, 35 (11)
  • [29] Endothelial deletion of protein tyrosine phosphatase-1B promotes angiogenesis and improves survival and heart function after pressure overload-induced cardiac hypertrophy in mice
    Gogiraju, R.
    Schroeter, M. R.
    Bochenek, M. L.
    Schaefer, K.
    EUROPEAN HEART JOURNAL, 2015, 36 : 362 - 362
  • [30] Response by Benitah et al to Letter Regarding Article, "Orai1 Channel Inhibition Preserves Left Ventricular Systolic Function and Normal Ca2+ Handling After Pressure Overload"
    Benitah, Jean-Pierre
    Beech, David J.
    Sabourin, Jessica
    CIRCULATION, 2020, 141 (22) : E839 - E840
123