Pulmonary disease severity in men with ΔF508 cystic fibrosis and residual chloride secretion (vol 353, pg 984, 1999)

被引:0
|
作者
Thomas, SR
机构
来源
LANCET | 1999年 / 353卷 / 9166期
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:1804 / 1804
页数:1
相关论文
共 44 条
  • [1] Pulmonary disease severity in men with ΔF508 cystic fibrosis and residual chloride secretion
    Thomas, SR
    Jaffe, A
    Geddes, DM
    Hodson, ME
    Alton, EWFW
    LANCET, 1999, 353 (9157): : 984 - 985
  • [2] Residual chloride secretion in intestinal tissue of ΔF508 homozygous twins and siblings with cystic fibrosis
    Bronsveld, I
    Mekus, F
    Bijman, J
    Ballmann, M
    Greipel, J
    Hundrieser, J
    Halley, DJJ
    Laabs, U
    Busche, R
    De Jonge, HR
    Tümmler, B
    Veeze, HJ
    GASTROENTEROLOGY, 2000, 119 (01) : 32 - 40
  • [3] ΔF508 heterozygosity in cystic fibrosis and susceptibility to asthma (vol 351, pg 1911, 1998)
    Dahl, M
    LANCET, 1998, 352 (9135): : 1230 - 1230
  • [4] Cystic Fibrosis Transmembrane Conductance Regulator ΔF508 Heterozygosity: A Spectrum of Disease Burden
    Banipal, J.
    Icard, B. L.
    Sweet, J. M.
    Loschner, A. L.
    AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2018, 197
  • [5] Pathophysiological basis of liver disease in cystic fibrosis employing a ΔF508 mouse model
    Freudenberg, Folke
    Broderick, Annemarie L.
    Yu, Bian B.
    Leonard, Monika R.
    Glickman, Jonathan N.
    Carey, Martin C.
    AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY, 2008, 294 (06): : G1411 - G1420
  • [6] Chloride conductance and genetic background modulate the cystic fibrosis phenotype of ΔF508 homozygous twins and siblings
    Bronsveld, I
    Mekus, F
    Bijman, J
    Ballmann, M
    de Jonge, HR
    Laabs, U
    Halley, DJ
    Ellemunter, H
    Mastella, G
    Thomas, S
    Veeze, HJ
    Tümmler, B
    JOURNAL OF CLINICAL INVESTIGATION, 2001, 108 (11): : 1705 - 1715
  • [7] The ΔF508 Mutation Causes CFTR Misprocessing and Cystic Fibrosis-Like Disease in Pigs
    Ostedgaard, Lynda S.
    Meyerholz, David K.
    Chen, Jeng-Haur
    Pezzulo, Alejandro A.
    Karp, Philip H.
    Rokhlina, Tatiana
    Ernst, Sarah E.
    Hanfland, Robert A.
    Reznikov, Leah R.
    Ludwig, Paula S.
    Rogan, Mark P.
    Davis, Greg J.
    Dohrn, Cassie L.
    Wohlford-Lenane, Christine
    Taft, Peter J.
    Rector, Michael V.
    Hornick, Emma
    Nassar, Boulos S.
    Samuel, Melissa
    Zhang, Yuping
    Richter, Sandra S.
    Uc, Aliye
    Shilyansky, Joel
    Prather, Randall S.
    McCray, Paul B., Jr.
    Zabner, Joseph
    Welsh, Michael J.
    Stoltz, David A.
    SCIENCE TRANSLATIONAL MEDICINE, 2011, 3 (74)
  • [8] Mild processing defect of porcine ΔF508-CFTR suggests that ΔF508 pigs may not develop cystic fibrosis disease
    Liu, Yanli
    Wang, Ying
    Jiang, Yong
    Zhu, Na
    Liang, Haitao
    Xu, Lina
    Feng, Xuechao
    Yang, Hong
    Ma, Tonghui
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2008, 373 (01) : 113 - 118
  • [9] ACID AND BASE SECRETION IN FRESHLY EXCISED NASAL TISSUE FROM CYSTIC FIBROSIS PATIENTS WITH ΔF508 MUTATION
    Cho, D.
    Illek, B.
    Hwang, P. H.
    Fischer, H.
    PEDIATRIC PULMONOLOGY, 2011, : 241 - 242
  • [10] Acid and base secretion in freshly excised nasal tissue from cystic fibrosis patients with ΔF508 mutation
    Cho, Do-Yeon
    Hwang, Peter H.
    Illek, Beate
    Fischer, Horst
    INTERNATIONAL FORUM OF ALLERGY & RHINOLOGY, 2011, 1 (02) : 123 - 127