Innate Defense against Fungal Pathogens

被引:63
作者
Drummond, Rebecca A. [1 ]
Gaffen, Sarah L. [2 ]
Hise, Amy G. [3 ,4 ]
Brown, Gordon D. [1 ]
机构
[1] Univ Aberdeen, Aberdeen Fungal Grp, Aberdeen AB25 2ZD, Scotland
[2] Univ Pittsburgh, Div Rheumatol & Clin Immunol, Dept Med, Pittsburgh, PA 15261 USA
[3] Case Western Reserve Univ, Sch Med, Dept Pathol, Cleveland, OH 44106 USA
[4] Louis Stokes Vet Affairs Med Ctr, Dept Med, Cleveland, OH 44106 USA
基金
美国国家卫生研究院; 英国惠康基金; 英国医学研究理事会;
关键词
C-TYPE LECTIN; T-CELL RESPONSES; BETA-GLUCAN RECEPTOR; EXTRACELLULAR TRAP FORMATION; ADAPTIVE IMMUNE-RESPONSES; CANDIDA-ALBICANS; HOST-DEFENSE; DENDRITIC CELLS; NALP3; INFLAMMASOME; ASPERGILLUS-FUMIGATUS;
D O I
10.1101/cshperspect.a019620
中图分类号
R-3 [医学研究方法]; R3 [基础医学];
学科分类号
1001 ;
摘要
Human fungal infections have been on the rise in recent years and proved increasingly difficult to treat as a result of the lack of diagnostics, effective antifungal therapies, and vaccines. Most pathogenic fungi do not cause disease unless there is a disturbance in immune homeostasis, which can be caused by modern medical interventions, disease-induced immunosuppression, and naturally occurring human mutations. The innate immune system is well equipped to recognize and destroy pathogenic fungi through specialized cells expressing a broad range of pattern recognition receptors (PRRs). This review will outline the cells and PRRs required for effective antifungal immunity, with a special focus on the major antifungal cytokine IL-17 and recently characterized antifungal inflammasomes.
引用
收藏
页码:1 / 19
页数:19
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