Premenopausal Osteoporosis

被引：34

作者：

Cohen, Adi ^{[1
]}

机构：

[1] Columbia Univ Coll Phys & Surg, Div Endocrinol, Dept Med, Med Ctr, PH8-864,630 West 168th St, New York, NY 10032 USA

来源：

ENDOCRINOLOGY AND METABOLISM CLINICS OF NORTH AMERICA | 2017年 / 46卷 / 01期

关键词：

Premenopausal women; Secondary causes of osteoporosis; Osteoporosis treatment; BONE-MINERAL DENSITY; GLUCOCORTICOID-INDUCED OSTEOPOROSIS; DEPOT MEDROXYPROGESTERONE ACETATE; RANDOMIZED CONTROLLED-TRIAL; LACTATION-ASSOCIATED OSTEOPOROSIS; YOUNG-ADULT WOMEN; IDIOPATHIC OSTEOPOROSIS; ANOREXIA-NERVOSA; POSTMENOPAUSAL WOMEN; CLINICAL CHARACTERISTICS;

D O I：

10.1016/j.ecl.2016.09.007

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Most premenopausal women with low trauma fracture(s) or low bone mineral density have a secondary cause of osteoporosis or bone loss. Where possible, treatment of the underlying cause should be the focus of management. Premenopausal women with an ongoing cause of bone loss and those who have had, or continue to have, low trauma fractures may require pharmacologic intervention. Clinical trials provide evidence of benefits of bisphosphonates and teriparatide for bone mineral density in several types of premenopausal osteoporosis, but studies are small and do not provide evidence regarding fracture risk reduction.

引用

页码：117 / +

页数：18

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