Critically ill Patients and Platelet-Derived Microparticles

[1] Cell-derived microparticles in haemostasis and vascular medicine [J]. THROMBOSIS AND HAEMOSTASIS, 2009, 101 (03) : 439 - 451

[2] Medical progress: The pathophysiology and treatment of sepsis. [J]. NEW ENGLAND JOURNAL OF MEDICINE, 2003, 348 (02) : 138 - 150

[3] Universal changes in biomarkers of coagulation and inflammation occur in patients with severe sepsis, regardless of causative micro-organism [ISRCTN74215569] [J]. Critical Care, 8 (2):

[4] Combination of thrombin-antithrombin complex, plasminogen activator inhibitor-1, and protein C activity for early identification of severe coagulopathy in initial phase of sepsis: a prospective observational study [J]. CRITICAL CARE, 2014, 18 (01)

[5] High shear stress can initiate both platelet aggregation and shedding of procoagulant containing microparticles [J]. BLOOD, 1996, 88 (09) : 3456 - 3464

[6] Clinical significance of procoagulant microparticles [J]. Journal of Intensive Care, 3 (1)

[7] The correlation between platelet activation markers and HMGB1 in patients with disseminated intravascular coagulation and hematologic malignancy [J]. PLATELETS, 2011, 22 (05) : 396 - 397

[8] Nomura S, 2009, J ATHEROSCLER THROMB, V16, P878

[9] Association of the Plasma Platelet-Derived Microparticles to Platelet Count Ratio with Hospital Mortality and Disseminated Intravascular Coagulopathy in Critically Ill Patients [J]. JOURNAL OF ATHEROSCLEROSIS AND THROMBOSIS, 2015, 22 (08) : 773 - 782

[10] Circulating angiopoietin-2 levels in the course of septic shock: relation with fluid balance, pulmonary dysfunction and mortality [J]. INTENSIVE CARE MEDICINE, 2009, 35 (09) : 1567 - 1574

[1] Cell-derived microparticles in haemostasis and vascular medicine [J]. THROMBOSIS AND HAEMOSTASIS, 2009, 101 (03) : 439 - 451

[2] Medical progress: The pathophysiology and treatment of sepsis. [J]. NEW ENGLAND JOURNAL OF MEDICINE, 2003, 348 (02) : 138 - 150

[3] Universal changes in biomarkers of coagulation and inflammation occur in patients with severe sepsis, regardless of causative micro-organism [ISRCTN74215569] [J]. Critical Care, 8 (2):

[4] Combination of thrombin-antithrombin complex, plasminogen activator inhibitor-1, and protein C activity for early identification of severe coagulopathy in initial phase of sepsis: a prospective observational study [J]. CRITICAL CARE, 2014, 18 (01)

[5] High shear stress can initiate both platelet aggregation and shedding of procoagulant containing microparticles [J]. BLOOD, 1996, 88 (09) : 3456 - 3464

[6] Clinical significance of procoagulant microparticles [J]. Journal of Intensive Care, 3 (1)

[7] The correlation between platelet activation markers and HMGB1 in patients with disseminated intravascular coagulation and hematologic malignancy [J]. PLATELETS, 2011, 22 (05) : 396 - 397

[8] Nomura S, 2009, J ATHEROSCLER THROMB, V16, P878

[9] Association of the Plasma Platelet-Derived Microparticles to Platelet Count Ratio with Hospital Mortality and Disseminated Intravascular Coagulopathy in Critically Ill Patients [J]. JOURNAL OF ATHEROSCLEROSIS AND THROMBOSIS, 2015, 22 (08) : 773 - 782

[10] Circulating angiopoietin-2 levels in the course of septic shock: relation with fluid balance, pulmonary dysfunction and mortality [J]. INTENSIVE CARE MEDICINE, 2009, 35 (09) : 1567 - 1574