MicroRNAs play big roles in modulating macrophages response toward mycobacteria infection

被引：24
作者：
Abdalla, Abualgasim Elgaili [1 ,2 ]
Duan, Xiangke [1 ]
Deng, Wanyan [1 ]
Zeng, Jie [1 ]
Xie, Jianping [1 ]
机构：
[1] Southwest Univ, Sch Life Sci,Minist Educ, Key Lab Ecoenvironm Gorges Reservoir Reg 3, Inst Modern Biopharmaceut,State Key Lab Breeding, Chongqing 400715, Peoples R China
[2] Omdurman Islamic Univ, Coll Med Lab Sci, Dept Clin Microbiol, Khartoum, Sudan
来源：
INFECTION GENETICS AND EVOLUTION | 2016年 / 45卷
关键词：
miRs; Mycobacterium tuberculosis; Macrophages; Autophagy; Apoptosis; Inflammatory; INFLAMMATORY RESPONSE; TUBERCULOSIS; AUTOPHAGY; EXPRESSION; INDUCTION; INHIBITION; MIR-155; CELLS; MONOCYTES; DEFENSE;
D O I：
10.1016/j.meegid.2016.09.023
中图分类号：
R51 [传染病];
学科分类号：
100401 ;
摘要：
Macrophages are crucial player in the defense against multiple intracellular pathogens. Mycobacterium tuberculosis, the causative agent of tuberculosis which inflicted around one third of global population, can replicate and persist within macrophages. MicroRNAs, endogenous, small noncoding RNA, can regulate the expression of macrophages genes required for appropriate signaling. Mycobacteria can manipulate the expression of macrophages microRNAs to subvert cell response for its survival and persistence. This review summarized the progress of microRNAs in mycobacterial pathogenesis. (C) 2016 Elsevier B.V. All rights reserved.
引用
收藏
页码：378 / 382
页数：5
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