Crambene induces pancreatic acinar cell apoptosis via the activation of mitochondrial pathway

被引:10
作者
Cao, Yang
Adhikari, Sharmila
Ang, Abel Damien
Clement, Marie Veronique
Wallig, Matthew
Bhatia, Madhav
机构
[1] Natl Univ Singapore, Dept Pharmacol, Yong Loo Lin Sch Med, Singapore 117597, Singapore
[2] Natl Univ Singapore, Dept Biochem, Singapore 117597, Singapore
[3] Univ Illinois, Dept Pathobiol, Urbana, IL USA
来源
AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY | 2006年 / 291卷 / 01期
关键词
caspases; cytochrome c; membrane potential;
D O I
10.1152/ajpgi.00520.2005
中图分类号
R57 [消化系及腹部疾病];
学科分类号
摘要
We investigated the apoptotic pathway activated by crambene (1-cyano-2-hydroxy-3-butene), a plant nitrile, on pancreatic acinar cells. As evidenced by annexin V-FITC staining, crambene treatment for 3 h induced the apoptosis but not necrosis of pancreatic acini. Caspase-3, -8, and -9 activities in acini treated with crambene were significantly higher than in untreated acini. Treatment with caspase-3, -8, and -9 inhibitors inhibited annexin V staining, as well as caspase-3 activity, pointing to an important role of these caspases in crambene-induced acinar cell apoptosis. The mitochondrial membrane potential was collapsed, and cytochrome c was released from the mitochondria in crambene-treated acini. Neither TNF-alpha nor Fas ligand levels were changed in pancreatic acinar cells after crambene treatment. These results provide evidence for the induction of pancreatic acinar cell apoptosis in vitro by crambene and suggest the involvement of mitochondrial pathway in pancreatic acinar cell apoptosis.
引用
收藏
页码:G95 / G101
页数:7
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