Pathologic Cardiac Hypertrophy, but not Physiologic Cardiac Growth, Induces O-GlcNAc Signaling

被引:0
|
作者
Brainard, Robert Eli
Ngoh, Gladys A.
Facundo, Heberty T. [1 ]
Stoller, Phillip M.
Jones, Steven P. [1 ]
机构
[1] Univ Louisville, Inst Mol Cardiol, Louisville, KY 40292 USA
来源
FASEB JOURNAL | 2011年 / 25卷
关键词
D O I
暂无
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
引用
收藏
页数:1
相关论文
共 50 条
  • [31] Assessment Of Physiologic And Pathologic Cardiac Hypertrophy By Two-dimensional Strain And Torsion Analysis
    Vitarelli, Antonio
    Bernardi, Mauro
    Placanica, Giuseppe
    Conde, Ysabel
    D'Orazio, Simona
    Stallato, Simona
    Battaglia, Daniela
    Caranci, Fiorella
    Continanza, Giovanna
    Dettod, Olga
    Placanica, Attilio
    Vitarelli, Massimo
    Capotosto, Lidia
    CIRCULATION, 2008, 118 (18) : S852 - S852
  • [32] Characterization of physiologic cardiac hypertrophy in mice
    Tsirka, AE
    Rogers, JH
    Muslin, AJ
    CIRCULATION, 2001, 104 (17) : 165 - 165
  • [33] Increased Cardiac O-GlcNAc Transferase and O-Glcnacase Association to Actin, Tropomyosin and MLC 1 in Diabetes: A Mechanism for O-GlcNAc Mediated Myofilament Calcium Desensitization
    Ramirez-Correa, Genaro A.
    Slawson, Chad
    Gao, Wei D.
    Hart, Gerald W.
    Murphy, Anne M.
    BIOPHYSICAL JOURNAL, 2011, 100 (03) : 451 - 451
  • [34] FGFR4 Is Required for Concentric Growth of Cardiac Myocytes during Physiologic Cardiac Hypertrophy
    Campos, Isaac
    Richter, Beatrice
    Thomas, Sarah Madison
    Czaya, Brian
    Yanucil, Christopher
    Kentrup, Dominik
    Fajol, Abul
    Li, Qing
    Secor, Stephen M.
    Faul, Christian
    JOURNAL OF CARDIOVASCULAR DEVELOPMENT AND DISEASE, 2024, 11 (10)
  • [35] Control of Nrf2 Signaling by O-GlcNAc
    Smith, T.
    Chen, P.
    Chi, J.
    Boyce, M.
    MOLECULAR BIOLOGY OF THE CELL, 2015, 26
  • [36] The role of O-GlcNAc signaling in the pathogenesis of diabetic retinopathy
    Semba, Richard D.
    Huang, Hu
    Lutty, Gerard A.
    Van Eyk, Jennifer E.
    Hart, Gerald W.
    PROTEOMICS CLINICAL APPLICATIONS, 2014, 8 (3-4) : 218 - 231
  • [37] O-GlcNAc modification, insulin signaling and diabetic complications
    Issad, T.
    Masson, E.
    Pagesy, P.
    DIABETES & METABOLISM, 2010, 36 (06) : 423 - 435
  • [38] Akt1 promotes physiologic, but antagonizes pathologic, cardiac growth
    DeBosch, BJ
    Lupu, T
    Muslin, A
    CIRCULATION, 2004, 110 (17) : 155 - 155
  • [39] O-GlcNAc signaling attenuates mitochondrial permeability transition
    Ngoh, Gladys Afor
    Jones, Steven P.
    FASEB JOURNAL, 2008, 22
  • [40] Roles for O-Glcnac Glycosylation in Transcription and Neuronal Signaling
    Hsieh-Wilson, Linda
    GLYCOBIOLOGY, 2009, 19 (11) : 1299 - 1299