Origins of Cystic Fibrosis Lung Disease

被引：100

作者：

Munder, Antje ^{[1
]}

Tuemmler, Burkhard ^{[1
]}

机构：

[1] Hannover Med Sch, Hannover, Germany

来源：

关键词：

D O I：

10.1056/NEJMc1502191

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1574 / 1574

页数：1

共 5 条

[1] Absence of the cystic fibrosis transmembrane regulator (Cftr) from myeloid-derived cells slows resolution of inflammation and infection
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[2] Dysfunctional CFTR Alters the Bactericidal Activity of Human Macrophages against Pseudomonas aeruginosa
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[3] Neutrophil-Mediated Phagocytic Host Defense Defect in Myeloid Cftr-Inactivated Mice
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[J]. PLOS ONE, 2014, 9 (09):
[4] Origins of Cystic Fibrosis Lung Disease
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[J]. NEW ENGLAND JOURNAL OF MEDICINE, 2015, 372 (04) : 351 - 362
[5] Optimal Complement-Mediated Phagocytosis of Pseudomonas aeruginosa by Monocytes Is Cystic Fibrosis Transmembrane Conductance Regulator-Dependent
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Speirs, Jennifer J.
Pals, D. J.
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[J]. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 2013, 49 (03) : 463 - 470