Toward a standard model for autophagosome biogenesis

被引：9

作者：

Cook, Annan S. I. ^{[1
,2
,3
]}

Hurley, James H. ^{[1
,2
,3
,4
,5
]}

机构：

[1] Univ Calif Berkeley, Grad Grp Biophys, Berkeley, CA 94720 USA

[2] Univ Calif Berkeley, Calif Inst Quantitat Biosci, Berkeley, CA 94720 USA

[3] Aligning Sci Parkinsons ASAP Collaborat Res Networ, Chevy Chase, MD 20815 USA

[4] Univ Calif Berkeley, Dept Mol & Cell Biol, Berkeley, CA 94720 USA

[5] Univ Calif Berkeley, Helen Wills Neurosci Inst, Berkeley, CA 94720 USA

来源：

JOURNAL OF CELL BIOLOGY | 2023年 / 222卷 / 07期

关键词：

D O I：

10.1083/jcb.202304011

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

Two papers in this issue resolve a long-standing obstacle to a "standard model" for autophagosome biogenesis in mammals. The first, Olivas et al. (2023. J. Cell Biol. ), uses biochemistry to confirm that the lipid scramblase ATG9A is a bona fide autophagosome component, while the second, Broadbent et al. (2023. J. Cell Biol. ), uses particle tracking to show that the dynamics of autophagy proteins are consistent with the concept.

引用

页数：3

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