Does MPK4/12-HT1 function as a CO2/bicarbonate sensor to regulate the stomatal conductance under high CO2 levels?

被引：0

作者：

Gahlowt, Priya ^{[1
]}

Tripathi, Durgesh Kumar ^{[2
]}

Singh, Samiksha ^{[3
]}

Gupta, Ravi ^{[4
]}

Singh, Vijay Pratap ^{[1
]}

机构：

[1] Univ Allahabad, CMP Degree Coll, Plant Physiol Lab, Dept Bot,Constituent Post Grad Coll, Prayagraj 211002, India

[2] Amity Univ Uttar Pradesh, Inst Organ Agr, Crop Nanobiol & Mol Stress Physiol Lab Am, Sect 125, Noida 201313, India

[3] Chhatrapati Shahu Ji Maharaj Univ, SN Sen BV Post Grad Coll, Dept Bot, Kanpur 208001, India

[4] Kookmin Univ, Coll Gen Educ, Seoul 02707, South Korea

来源：

PLANT CELL REPORTS | 2023年 / 42卷 / 12期

关键词：

KINASE;

D O I：

10.1007/s00299-023-03077-8

中图分类号：

Q94 [植物学];

学科分类号：

071001 ;

摘要：

Key message Recently, a HT1 protein has been identified which causes continuous opening of stomata because of its kinase activity. However, reversible interaction between MAP4/12 and HT1 protein acts as a CO2/bicarbonate sensor and causes the closing of stomata by inhibiting HT1 kinase activity.

引用

页码：2043 / 2045

页数：3

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