2型糖尿病患者的非糖尿病一级亲属的脂代谢紊乱

[1] Decresedinsulinactivationofglycogen synthasein skeletal muscles in young nonobese Caucasianfirstdegreerelativesofpatients with noninsulindependentdiabetes mellitus〔J〕. Vaag Aetal. The Journal of Clinical Investigation . 1992

[2] Longterm exposure ofrat pancreaticisletstofatty acidsinhibitsglucoseinducedinsulinsecretion and biosynthesisthrough a glucosefatty acid cycle〔J〕. Zhou YP,Grill VE. The Journal of Clinical Investigation . 1994

[3] Insulin resistanceand hyperinsulinemiain individuals with small,dense,low density lipoproteinparticles 〔J〕. Reaven GMetal. The Journal of Clinical Investigation . 1993

[4] Betacelldysfunctioninfirstdegreerelativesofpatients with noninsulindependentdiabetesmellitus〔J〕. Fernandez CMetal. Diabetic Medicine . 1996

[5] Changesoflipolyticenzymescluster withinsulinresistancesyndrome〔J〕. Knudsen Petal. Diabetologia . 1995

[6] Therelationship betweeninsulinsensitivityandthefattyacidcompositionofskeletal musclephospholipids〔J〕. Borman Metal. The New England Journal of Medicine . 1993

[7] Fat metabolism in human obesity 〔J〕. CampbellPJetal. American Journal of Physiology . 1994

[8] Dyslipidemia and hyperinsulinemiain normoglycemicobese relatives of patients with noninsulindependentdiabetes mellitus 〔J〕. Florez Het al. Investigación Clínica . 1995

[9] Tumornecrosisfactorαinducedinsulinresistancein3T3L1adipocytesisaccompanied byalossofinsulin receptor substrate1 and GLUT4 expressionwithoutalossofinsulin receptormediated signaltransduction 〔J〕. StephensJMetal. Journal of Biological Chemistry . 1997

[10] Tissuespecificregulationoflipoproteinlipase activity by insulin/glucose in normalweight humans〔J〕. Farese RVetal. Metabolism . 1991

[1] Decresedinsulinactivationofglycogen synthasein skeletal muscles in young nonobese Caucasianfirstdegreerelativesofpatients with noninsulindependentdiabetes mellitus〔J〕. Vaag Aetal. The Journal of Clinical Investigation . 1992

[2] Longterm exposure ofrat pancreaticisletstofatty acidsinhibitsglucoseinducedinsulinsecretion and biosynthesisthrough a glucosefatty acid cycle〔J〕. Zhou YP,Grill VE. The Journal of Clinical Investigation . 1994

[3] Insulin resistanceand hyperinsulinemiain individuals with small,dense,low density lipoproteinparticles 〔J〕. Reaven GMetal. The Journal of Clinical Investigation . 1993

[4] Betacelldysfunctioninfirstdegreerelativesofpatients with noninsulindependentdiabetesmellitus〔J〕. Fernandez CMetal. Diabetic Medicine . 1996

[5] Changesoflipolyticenzymescluster withinsulinresistancesyndrome〔J〕. Knudsen Petal. Diabetologia . 1995

[6] Therelationship betweeninsulinsensitivityandthefattyacidcompositionofskeletal musclephospholipids〔J〕. Borman Metal. The New England Journal of Medicine . 1993

[7] Fat metabolism in human obesity 〔J〕. CampbellPJetal. American Journal of Physiology . 1994

[8] Dyslipidemia and hyperinsulinemiain normoglycemicobese relatives of patients with noninsulindependentdiabetes mellitus 〔J〕. Florez Het al. Investigación Clínica . 1995

[9] Tumornecrosisfactorαinducedinsulinresistancein3T3L1adipocytesisaccompanied byalossofinsulin receptor substrate1 and GLUT4 expressionwithoutalossofinsulin receptormediated signaltransduction 〔J〕. StephensJMetal. Journal of Biological Chemistry . 1997

[10] Tissuespecificregulationoflipoproteinlipase activity by insulin/glucose in normalweight humans〔J〕. Farese RVetal. Metabolism . 1991