p19ARF与肿瘤

[1] Impaired pro-cessing of DNA photoproducts and ultraviolet hypermutability withloss of p16INK4a or p19ARF. Sarkar-Agrawal P,Vergilis I,Sharpless NE,et al. Journal of the National Cancer Institute . 2004

[2] Targeting of C-terminal bindingprotein(CtBP)by ARF results in p53-independent apoptosis. Paliwal S,Pande S,Kovi RC,et al. Molecular and Cellular Biology . 2006

[3] p16Ink4a or p19Arfloss contributes to Tall-induced leukemogenesis in mice. Shank-Calvo JA,Draheim K,Bhasin M,et al. Oncegene . 2006

[4] p19Arf suppressesgrowth,progression,and metastasis of Hras-driven carcinomasthrough p53-dependent and-independent pathways. Kelly-Spratt KS,Gurley KE,Yasui Y,et al. PLoS Bi-ol . 2004

[5] E2F1 suppresses skin car-cinogenesis via the ARF-p53 pathway. Russell JL,Weaks RL,Berton TR,et al. Oncegene . 2006

[6] ARF-BP1/Mule is a critical mediatorof the ARF tumor suppressor. Chen D,Kon N,Li M,et al. Cell . 2005

[7] The fibroblast growth factor-2 is notessential for melanoma formation in a transgenic mouse model. Ackermann J,Beermann F. Pigment Cell Research . 2005

[8] Methylationstatusanalysis of cell cycle regulatory genes(p16 INK4A,p15 INK4B,p21Wafl/Cipl,p27Kipl and p73)in natural killer cell disorders. Kawamata N,Inagaki N,Mizumura S,et al. Eur J Haem atol . 2005

[9] Cooperative signals governing ARF-mdm2 interaction and nucleolar localization of the complex.Mol. J.D.Weber,M.L.Kuo B,et al. The Journal of Cell Biology . 2000

[10] Regulation of Myc function by ARF:Check-point for Myc-Induced Oncogenesis. Sarkar D,Fisher PB. Cancer Biology and Therapy . 2006

[1] Impaired pro-cessing of DNA photoproducts and ultraviolet hypermutability withloss of p16INK4a or p19ARF. Sarkar-Agrawal P,Vergilis I,Sharpless NE,et al. Journal of the National Cancer Institute . 2004

[2] Targeting of C-terminal bindingprotein(CtBP)by ARF results in p53-independent apoptosis. Paliwal S,Pande S,Kovi RC,et al. Molecular and Cellular Biology . 2006

[3] p16Ink4a or p19Arfloss contributes to Tall-induced leukemogenesis in mice. Shank-Calvo JA,Draheim K,Bhasin M,et al. Oncegene . 2006

[4] p19Arf suppressesgrowth,progression,and metastasis of Hras-driven carcinomasthrough p53-dependent and-independent pathways. Kelly-Spratt KS,Gurley KE,Yasui Y,et al. PLoS Bi-ol . 2004

[5] E2F1 suppresses skin car-cinogenesis via the ARF-p53 pathway. Russell JL,Weaks RL,Berton TR,et al. Oncegene . 2006

[6] ARF-BP1/Mule is a critical mediatorof the ARF tumor suppressor. Chen D,Kon N,Li M,et al. Cell . 2005

[7] The fibroblast growth factor-2 is notessential for melanoma formation in a transgenic mouse model. Ackermann J,Beermann F. Pigment Cell Research . 2005

[8] Methylationstatusanalysis of cell cycle regulatory genes(p16 INK4A,p15 INK4B,p21Wafl/Cipl,p27Kipl and p73)in natural killer cell disorders. Kawamata N,Inagaki N,Mizumura S,et al. Eur J Haem atol . 2005

[9] Cooperative signals governing ARF-mdm2 interaction and nucleolar localization of the complex.Mol. J.D.Weber,M.L.Kuo B,et al. The Journal of Cell Biology . 2000

[10] Regulation of Myc function by ARF:Check-point for Myc-Induced Oncogenesis. Sarkar D,Fisher PB. Cancer Biology and Therapy . 2006