Leptin-p38MAPK pathway mediates high glucose-induced injury in H9c2 cardiac cells

被引：0
|
作者：
徐文明 [1 ]
机构：
[1] Dept Intern Med,Huangpu Div,1st Affil Hosp, Sun Yat-Sen Univ
来源：
China Medical Abstracts(Internal Medicine) | 2014年 / 31卷 / 03期
关键词：
MAPK; Leptin-p38MAPK pathway mediates high glucose-induced injury in H9c2 cardiac cells;
D O I：
暂无
中图分类号：
R542.2 [心肌疾病];
学科分类号：
1002 ; 100201 ;
摘要：
Objective To investigate the role of leptin-p38 mitogen-activated protein kinase(MAPK)pathway in high glucose(HG)-induced injury in H9c2 cardiac cells.Methods H9c2 cardiac cells were treated with HG to establish a model of cellular injury induced by HG.Cell viability was tested by cell counter kit 8(CCK-8).The intracellular level of reactive oxygen species(ROS)was
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页码：163 / 164
页数：2
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