The Role of Neutrophil Extracellular Traps in Atherosclerosis: From the Molecular to the Clinical Level

被引:0
作者
Yuan, Yongfang [1 ]
Sun, Changxin [2 ]
Liu, Xinyi [2 ]
Hu, Lanqing [1 ]
Wang, Zeping [1 ]
Li, Xiaoya [1 ]
Zhang, Jingyi [2 ]
Li, Dexiu [1 ]
Zhang, Xiaonan [1 ]
Wu, Min [3 ]
Liu, Longtao [1 ]
机构
[1] China Acad Chinese Med Sci, Xiyuan Hosp, Natl Clin Res Ctr Chinese Med Cardiol, State Key Lab Tradit Chinese Med Syndrome, Beijing, Peoples R China
[2] Beijing Univ Chinese Med, Beijing, Peoples R China
[3] China Acad Chinese Med Sci, Guangan Men Hosp, Beijing, Peoples R China
基金
中国国家自然科学基金; 北京市自然科学基金;
关键词
atherosclerosis; neutrophil traps; inflammation; lipid metabolism; therapeutic strategies; ENDOTHELIAL DYSFUNCTION; MESENCHYMAL TRANSITION; THROMBIN GENERATION; CATHEPSIN-G; MYELOPEROXIDASE; HISTONES; NETOSIS; ACTIVATION; ELASTASE; DNA;
D O I
10.2147/JIR.S507330
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Atherosclerosis is a chronic inflammatory condition that is typified by the deposition of lipids and the subsequent inflammation of medium and large arteries. Neutrophil extracellular traps (NETs) are fibrous meshworks of DNA, histones, and granzymes expelled by activated neutrophils in response to a variety of pathogenic conditions. In addition to their role in pathogen eradication, NETs have been demonstrated to play a pivotal role in the development of atherosclerosis. This article presents a review of the bidirectional interactions in which atherosclerosis-related risk factors stimulate the formation of NETs, which in turn support disease progression. This article emphasizes the involvement of NETs in the various stages of atherogenesis and development, influencing multiple factors such as the vascular endothelium, platelets, the inflammatory milieu, and lipid metabolism. The findings of this study offer new insights and avenues for further investigation into the processes underlying the formation and regulation of the vascular inflammatory microenvironment in atherosclerosis. Finally, potential targeted therapeutic strategies for NETs are discussed to facilitate their progression to clinical practice (Graphical Abstract).
引用
收藏
页码:4421 / 4433
页数:13
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