Ezrin, a novel marker of ependymal cells, can be used to demonstrate their proliferation regulation after spinal cord injury in mice

被引:0
作者
Zhang, Lin [1 ,2 ,4 ]
Xu, Yao-Mei [1 ,2 ]
Bian, Ming-Ming [1 ,2 ]
Yan, Hua-Zheng [1 ,2 ]
Gao, Jian-Xiong [1 ,2 ]
Bao, Qian-Hui [1 ,2 ]
Chen, Yu-Qing [1 ,2 ]
Ding, Shu-Qin [1 ]
Wang, Rui [2 ]
Zhang, Nan [2 ]
Hu, Jian-Guo [1 ,2 ,5 ]
Lu, He-Zuo [1 ,2 ,3 ,5 ]
机构
[1] Bengbu Med Univ, Affiliated Hosp 1, Clin Lab, Bengbu 233004, Anhui, Peoples R China
[2] Bengbu Med Univ, Affiliated Hosp 1, Anhui Key Lab Tissue Transplantat, Bengbu 233004, Anhui, Peoples R China
[3] Bengbu Med Univ, Anhui Prov Key Lab Immunol Chron Dis, Bengbu 233030, Anhui, Peoples R China
[4] Bengbu Med Univ, Sch Life Sci, Bengbu 233030, Anhui, Peoples R China
[5] Bengbu Med Univ, Anhui Prov Key Lab Basic & Translat Res Inflammat, Bengbu 233030, Anhui, Peoples R China
基金
中国国家自然科学基金;
关键词
Spinal cord injury; Stem cell; Ependyma; Central canal; Single cell genomics; Ezrin; Guanosine monophosphate reductase; NEURAL STEM-CELLS; GENE-EXPRESSION; GROWTH-FACTOR; ATF3; DAMAGE; BRAIN; DIFFERENTIATION; ASTROCYTES; PROTEINS; REVEALS;
D O I
10.1016/j.nbd.2024.106746
中图分类号
Q189 [神经科学];
学科分类号
071006 ;
摘要
Ependymal cells (EpCs), as a potential stem cell niche, have gained interest for their potential in vivo stem cell therapy for spinal cord injury (SCI). Heterogeneity of spinal EpCs may contribute to differences in the ability of spinal EpCs to proliferate, differentiate and transition after injury, while there is limited understanding of the regulation of these events. Our research found that ezrin (Ezr) was expressed highly in EpCs of the spinal cord, and its upregulation rapidly occurred after injury (6 h). It remained consistently highly expressed in proliferating EpCs, this occurs before pathological accumulation of it occurs in other glial and immune-related cells. Differential expression of Ezr, Arg3, Pvalb, Ccnd1, and Gmpr characterized distinct responses of EpCs to injury activity. Also, we uncovered the dynamic regulatory behavior of immature EpCs after injury. In contrast to constitutive expression in parenchymal tissues, injury factors upregulated guanosine monophosphate reductase (Gmpr) in arrested EpCs, unveiling a distinctive mechanism to regulate proliferation in EpCs following spinal cord injury.
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页数:17
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