NLRP3: More than an Inflammasome?

被引:1
作者
Pennings, Gabrielle J. [1 ]
机构
[1] Univ Sydney, ANZAC Res Inst Sydney Local Hlth Dist, Concord, NSW, Australia
关键词
PHARMACOLOGICAL INHIBITION; ACTIVATION;
D O I
10.1055/a-2413-4672
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:1114 / 1116
页数:3
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共 21 条
  • [11] PYPAF1, a PYRIN-containing Apaf1-like protein that assembles with ASC and regulates activation of NF-κB
    Manji, GA
    Wang, L
    Geddes, BJ
    Brown, M
    Merriam, S
    Al-Garawi, A
    Mak, S
    Lora, JM
    Briskin, M
    Jurman, M
    Cao, J
    DiStefano, PS
    Bertin, J
    [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (13) : 11570 - 11575
  • [12] The inflammasome:: A molecular platform triggering activation of inflammatory caspases and processing of proIL-β
    Martinon, F
    Burns, K
    Tschopp, J
    [J]. MOLECULAR CELL, 2002, 10 (02) : 417 - 426
  • [13] NLRP3 inflammasome signalling in Alzheimer's disease
    McManus, Roisin M.
    Latz, Eicke
    [J]. NEUROPHARMACOLOGY, 2024, 252
  • [14] The NLRP3 inflammasome and bruton's tyrosine kinase in platelets co-regulate platelet activation, aggregation, and in vitro thrombus formation
    Murthy, Pranav
    Durco, Filip
    Miller-Ocuin, Jennifer L.
    Takedai, Teiko
    Shankar, Shruthi
    Liang, Xiaoyan
    Liu, Xiao
    Cui, Xiangdong
    Sachdev, Ulka
    Rath, Dominik
    Lotze, Michael T.
    Zeh, Herbert J., III
    Gawaz, Meinrad
    Weber, Alexander N.
    Vogel, Sebastian
    [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2017, 483 (01) : 230 - 236
  • [15] Rapid Release of Interleukin-1β from Human Platelets Is Independent of NLRP3 and Caspase
    Pennings, Gabrielle J.
    Reddel, Caroline J.
    Traini, Mathew
    Lam, Magdalena
    Kockx, Maaike
    Chen, Vivien M.
    Kritharides, Leonard
    [J]. THROMBOSIS AND HAEMOSTASIS, 2022, 122 (04) : 517 - 528
  • [16] NLRP3 regulates platelet integrin αIIbβ3 outside-in signaling, hemostasis and arterial thrombosis
    Qiao, Jianlin
    Wu, Xiaoqing
    Luo, Qi
    Wei, Guangyu
    Xu, Mengdi
    Wu, Yulu
    Liu, Yun
    Li, Xiaoqian
    Zi, Jie
    Ju, Wen
    Fu, Lin
    Chen, Chong
    Wu, Qingyun
    Zhu, Shengyun
    Qi, Kunming
    Li, Depeng
    Li, Zhenyu
    Andrews, Robert K.
    Zeng, Lingyu
    Gardiner, Elizabeth E.
    Xu, Kailin
    [J]. HAEMATOLOGICA, 2018, 103 (09) : 1568 - 1576
  • [17] Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
    Ridker, P. M.
    Everett, B. M.
    Thuren, T.
    MacFadyen, J. G.
    Chang, W. H.
    Ballantyne, C.
    Fonseca, F.
    Nicolau, J.
    Koenig, W.
    Anker, S. D.
    Kastelein, J. J. P.
    Cornel, J. H.
    Pais, P.
    Pella, D.
    Genest, J.
    Cifkova, R.
    Lorenzatti, A.
    Forster, T.
    Kobalava, Z.
    Vida-Simiti, L.
    Flather, M.
    Shimokawa, H.
    Ogawa, H.
    Dellborg, M.
    Rossi, P. R. F.
    Troquay, R. P. T.
    Libby, P.
    Glynn, R. J.
    [J]. NEW ENGLAND JOURNAL OF MEDICINE, 2017, 377 (12) : 1119 - 1131
  • [18] Gasdermin D-dependent platelet pyroptosis exacerbates NET formation and inflammation in severe sepsis
    Su, Meiling
    Chen, Chaofei
    Li, Shaoying
    Li, Musheng
    Zeng, Zhi
    Zhang, Yuan
    Xia, Luoxing
    Li, Xiuzhen
    Zheng, Dezhong
    Lin, Qiqi
    Fan, Xuejiao
    Wen, Ying
    Liu, Yingying
    Chen, Feiyan
    Luo, Wei
    Bu, Yun
    Qin, Jinhong
    Guo, Manli
    Qiu, Miaoyun
    Sun, Lei
    Liu, Renjing
    Wang, Ping
    Hwa, John
    Tang, Wai Ho
    [J]. NATURE CARDIOVASCULAR RESEARCH, 2022, 1 (08): : 732 - +
  • [19] The role of the NLRP3 inflammasome and pyroptosis in cardiovascular diseases
    Toldo, Stefano
    Abbate, Antonio
    [J]. NATURE REVIEWS CARDIOLOGY, 2024, 21 (04) : 219 - 237
  • [20] TLR4-dependent upregulation of the platelet NLRP3 inflammasome promotes platelet aggregation in a murine model of hindlimb ischemia
    Vogel, Sebastian
    Murthy, Pranav
    Cui, Xiangdong
    Lotze, Michael T.
    Zeh, Herbert J., III
    Sachdev, Ulka
    [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2019, 508 (02) : 614 - 619