MRPL44 Plays an Important Role in Cardiac Hypertrophy and Function

被引：0
|
作者：
Okamoto, Ryuji
Ye, Zhe
Ito, Rie
Moriwaki, Keishi
Ito, Hiromasa
Kimena, Lupiya
Ida, Mizuki
Dohi, Kaoru
机构：
来源：
CIRCULATION | 2024年 / 150卷
关键词：
D O I：
10.1161/circ.150.suppl_1.4123630
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
4123630
引用
收藏
页数：2
相关论文
共 50 条
  • [1] A Role for the Mitochondrial Protein Mrpl44 in Maintaining OXPHOS Capacity
    Yeo, Janet H. C.
    Skinner, Jarrod P. J.
    Bird, Matthew J.
    Formosa, Luke E.
    Zhang, Jian-Guo
    Kluck, Ruth M.
    Belz, Gabrielle T.
    Chong, Mark M. W.
    PLOS ONE, 2015, 10 (07):
  • [2] Gap Junction Protein Beta 4 Plays an Important Role In Cardiac Hypertrophy and Cardiac Function in Human and Zebrafish
    Okamoto, Ryuji
    Goto, Itaru
    Nishimura, Yuhei
    Kobayashi, Issei
    Hashizume, Ryotaro
    Dohi, Kaoru
    Yamada, Norikazu
    Tanaka, Toshio
    Ito, Masaaki
    CIRCULATION, 2016, 134
  • [3] Gap Junction Protein Beta 4 Plays an Important Role In Cardiac Hypertrophy and Cardiac Function in Human and Zebrafish
    Okamoto, Ryuji
    Goto, Itaru
    Nishimura, Yuhei
    Kobayashi, Issei
    Hashizume, Ryotaro
    Dohi, Kaoru
    Yamada, Norikazu
    Tanaka, Toshio
    Ito, Masaaki
    CIRCULATION, 2016, 134
  • [4] Elongator Protein 2 Plays an Important Role in Cardiac Hypertrophy In Vitro and In Vivo
    Okamoto, Ryuji
    Goto, Itaru
    Kobayashi, Issei
    Shimojo, Naoshi
    Dohi, Kaoru
    Nakamura, Mashio
    Ito, Masaaki
    JOURNAL OF CARDIAC FAILURE, 2014, 20 (10) : S188 - S188
  • [5] Pathogenic variants in MRPL44 cause infantile cardiomyopathy due to a mitochondrial translation defect
    Friederich, Marisa W.
    Geddes, Gabrielle C.
    Wortmann, Saskia B.
    Punnoose, Ann
    Wartchow, Eric
    Knight, Kaz M.
    Prokisch, Holger
    Creadon-Swindell, Geralyn
    Mayr, Johannes A.
    Van Hove, Johan L. K.
    MOLECULAR GENETICS AND METABOLISM, 2021, 133 (04) : 362 - 371
  • [6] MRPL44 mutations cause a slowly progressive multisystem disease with childhood-onset hypertrophic cardiomyopathy
    Distelmaier, Felix
    Haack, Tobias B.
    Catarino, Claudia B.
    Gallenmueller, Constanze
    Rodenburg, Richard J.
    Strom, Tim M.
    Baertling, Fabian
    Meitinger, Thomas
    Mayatepek, Ertan
    Prokisch, Holger
    Klopstock, Thomas
    NEUROGENETICS, 2015, 16 (04) : 319 - 323
  • [7] MRPL44 mutations cause a slowly progressive multisystem disease with childhood-onset hypertrophic cardiomyopathy
    Felix Distelmaier
    Tobias B. Haack
    Claudia B. Catarino
    Constanze Gallenmüller
    Richard J. Rodenburg
    Tim M. Strom
    Fabian Baertling
    Thomas Meitinger
    Ertan Mayatepek
    Holger Prokisch
    Thomas Klopstock
    neurogenetics, 2015, 16 : 319 - 323
  • [8] Thyroid Hormone Plays an Important Role in Cardiac Function: From Bench to Bedside
    Yamakawa, Hiroyuki
    Kato, Tomoko S.
    Noh, Jaeduk Yoshimura
    Yuasa, Shinsuke
    Kawamura, Akio
    Fukuda, Keiichi
    Aizawa, Yoshiyasu
    FRONTIERS IN PHYSIOLOGY, 2021, 12
  • [9] MRPL44 haploinsufficient mouse spontaneously develops hypertrophic cardiomyopathy and shows systolic dysfunction after aldosterone treatment
    Okamoto, R.
    Ye, Z.
    Ito, R.
    Moriwaki, K.
    Ito, H.
    Kimena, L.
    Ichikawa, M.
    Dohi, K.
    EUROPEAN HEART JOURNAL, 2024, 45
  • [10] Apelin-APJ system plays an important role in preventing catecholamine-induced cardiac hypertrophy
    Ichihara, N.
    Minamisawa, S.
    Hashimoto, T.
    Uchino, K.
    Fukamizu, A.
    Ishikawa, Y.
    Umemura, S.
    JOURNAL OF HYPERTENSION, 2008, 26 : S263 - S263
12345