机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Wang Lin
[1
]
Cui Ying
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Cui Ying
[1
]
Han Bingyu
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Han Bingyu
[1
]
Du Yitong
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Du Yitong
[1
]
Salewala Kenish Sirajbhai
论文数: 0引用数: 0
h-index: 0
机构:
International School, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Salewala Kenish Sirajbhai
[2
]
Wang Shiya
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Wang Shiya
[1
]
Zhao Wenlu
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Zhao Wenlu
[1
]
Zhang Hongxin
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Zhang Hongxin
[1
]
Wang Sichen
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Wang Sichen
[1
]
Xu Xinran
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Xu Xinran
[1
]
Ma Jianpeng
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Ma Jianpeng
[1
]
Zhu Yan
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Zhu Yan
[1
]
Tuo Houzhen
论文数: 0引用数: 0
h-index: 0
机构:
Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, ChinaDepartment of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
Tuo Houzhen
[1
]
机构:
[1] Department of Neurology, Beijing Friendship Hospital, Capital Medical University, Beijing, China
[2] International School, Capital Medical University, Beijing, China
Parkinson’s disease;
Microbiota-gut-brain axis;
Gut microbiota;
Neuroinflammation;
Metabolism;
D O I:
暂无
中图分类号:
R742.5 [震颤麻痹综合征];
学科分类号:
1002 ;
摘要:
Emerging evidence suggests that dysbiosis of the gut microbiota is associated with the pathogenesis of Parkinson’s disease (PD), a prevalent neurodegenerative disorder. The microbiota-gut-brain axis plays a crucial role in the development and progression of PD, and numerous studies have demonstrated the potential therapeutic benefits of modulations in the intestinal microbiota. This review provides insights into the characterization of the gut microbiota in patients with PD and highlights associations with clinical symptoms and underlying mechanisms. The discussion underscores the increased influence of the gut microbiota in the pathogenesis of PD. While the relationship is not fully elucidated, existing research demonstrates a strong correlation between changes in the composition of gut microbiota and disease development, and further investigation is warranted to explain the specific underlying mechanisms.