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- [6] CHARACTERIZATION OF FUNCTIONAL DOMAINS OF THE TENASCIN-R (RESTRICTIN) POLYPEPTIDE - CELL ATTACHMENT SITE, BINDING WITH F11, AND ENHANCEMENT OF F11-MEDIATED NEURITE OUTGROWTH BY TENASCIN-RJOURNAL OF CELL BIOLOGY, 1995, 130 (02): : 473 - 484NORENBERG, UHUBERT, MBRUMMENDORF, TTARNOK, ARATHJEN, FG
- [7] Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapsesScientific Reports, 9Christine GottschlingDavid WegrzynBernd DeneckeAndreas Faissner
- [8] Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapsesSCIENTIFIC REPORTS, 2019, 9 (1)Gottschling, ChristineWegrzyn, DavidDenecke, BerndFaissner, Andreas
- [9] Opposite impacts of tenascin-C and tenascin-R deficiency in mice on the functional outcome of facial nerve repairEUROPEAN JOURNAL OF NEUROSCIENCE, 2005, 22 (09) : 2171 - 2179Guntinas-Lichius, OAngelov, DNMorellini, FLenzen, MSkouras, ESchachner, MIrintchev, A
- [10] Involvement of chondroitin sulfates on brain-derived tenascin-R in carbohydrate-dependent interactions with fibronectin and tenascin-CBRAIN RESEARCH, 2000, 863 (1-2) : 42 - 51Probstmeier, RBraunewell, KHPesheva, P