Ginsenoside compound K attenuates ox-LDL-mediated macrophage inflammation and foam cell formation via autophagy induction and modulating NF-kB, p38, and JNK MAPK signaling (vol 11, 567238, 2020)

被引:0
作者
Lu, Shan [1 ,2 ,3 ,4 ,5 ]
Luo, Yun [1 ,2 ,3 ,4 ,5 ]
Sun, GuiBo [1 ,2 ,3 ,4 ,5 ]
Sun, XiaoBo [1 ,2 ,3 ,4 ,5 ]
机构
[1] Peking Union Med Coll & Chinese Acad Med Sci, Inst Med Plant Dev, Beijing, Peoples R China
[2] Inst Med Plant Dev, Beijing Key Lab Innovat Drug Discovery Tradit Chin, Beijing, Peoples R China
[3] Minist Educ, Key Lab Bioact Subst & Resource Utilizat Chinese H, Beijing, Peoples R China
[4] State Adm Tradit Chinese Med, Key Lab Efficacy Evaluat Chinese Med Glyeolipid Me, Beijing, Peoples R China
[5] Chinese Acad Med Sci, Key Lab New Drug Discovery Based Class Chinese Med, Beijing, Peoples R China
关键词
atherosclerosis; Ginsenoside compound K; inflammation; autophagy; macrophage;
D O I
10.3389/fphar.2024.1452605
中图分类号
R9 [药学];
学科分类号
1007 ;
摘要
引用
收藏
页数:2
相关论文
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  • [1] Ginsenoside Compound K Attenuates Ox-LDL-Mediated Macrophage Inflammation and Foam Cell FormationviaAutophagy Induction and Modulating NF-κB, p38, and JNK MAPK Signaling
    Lu, Shan
    Luo, Yun
    Sun, GuiBo
    Sun, XiaoBo
    [J]. FRONTIERS IN PHARMACOLOGY, 2020, 11