ACTIVE MYOCARDIAL DAMAGE IN HYPERTHYROIDISM - A CONCURRENT MECHANISM OF HEART-FAILURE REVERSED BY TREATMENT

被引：14

作者：

MARTI, V ^{[1
]}

BALLESTER, M ^{[1
]}

OBRADOR, D ^{[1
]}

MOYA, C ^{[1
]}

CARRIO, I ^{[1
]}

PONSLLADO, G ^{[1
]}

机构：

[1] HOSP SANTA CRUZ & SAN PABLO,NUCL MED UNIT,E-08025 BARCELONA,SPAIN

来源：

EUROPEAN HEART JOURNAL | 1995年 / 16卷 / 07期

关键词：

HYPERTHYROIDISM; MYOCARDIAL DAMAGE; MONOCLONAL ANTIMYOSIN ANTIBODIES; HEART FAILURE;

D O I：

10.1093/oxfordjournals.eurheartj.a061014

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

A patient with hyperthyroidism, heart failure, and depressed ejection fraction, in whom myocardial damage was evidenced by In-111-labelled monoclonal antimyosin antibodies, is presented. Both myocardial damage and left ventricular dysfunction disappeared after antithyroid therapy. This suggests that, in addition to volume overload, heart failure in hyperthyroidism could be also due to the presence of potentially reversible concurrent myocardial damage.

引用

页码：1014 / 1016

页数：3

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