CEREBRAL AMYLOID IN MICE WITH CREUTZFELDT-JAKOB DISEASE IS INFLUENCED BY THE STRAIN OF THE INFECTIOUS AGENT

被引：6

作者：

KITAMOTO, T

YI, R

MOHRI, S

TATEISHI, J

机构：

[1] KYUSHU UNIV 60,FAC MED,INST NEUROL,DEPT NEUROPATHOL,HIGASHI KU,FUKUOKA 812,JAPAN

[2] KYUSHU UNIV 60,CTR LAB ANIM,FUKUOKA 812,JAPAN

来源：

BRAIN RESEARCH | 1990年 / 508卷 / 01期

基金：

日本科学技术振兴机构;

关键词：

Agent strain; Amyloid; Creutzfeldt-Jakob disease; Infectious agent; Mouse; Prion protein;

D O I：

10.1016/0006-8993(90)91132-Z

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

We examined Fukuoka-1 and Fukuoka-2 mouse-adapted Creutzfeldt-Jakob disease strains. Mice infected with the Fukuoka-2 strain have a higher incidence of kuru plaques, a higher concentration of proteinase-resistant prion protein, and a higher infectivity titer than do mice with the Fukuoka-1 strain. Thus, it must be kept in mind that there is a difference in the strain of the infectious agent in murine Creutzfeldt-Jakob disease. © 1990.

引用

页码：165 / 167

页数：3

共 50 条

[31] Creutzfeldt-Jakob disease in the elderly
deSilva, R
Findlay, C
Awad, I
HarriesJones, R
Knight, R
Will, R
POSTGRADUATE MEDICAL JOURNAL, 1997, 73 (863) : 557 - 559
[32] Variant Creutzfeldt-Jakob disease
Ironside, J. W.
HAEMOPHILIA, 2010, 16 : 175 - 180
[33] Prion Strain Characterization of a Novel Subtype of Creutzfeldt-Jakob Disease
Galeno, Roberta
Di Bari, Michele Angelo
Nonno, Romolo
Cardone, Franco
Sbriccoli, Marco
Graziano, Silvia
Ingrosso, Loredana
Fiorini, Michele
Valanzano, Angelina
Pasini, Giulia
Poleggi, Anna
Vinci, Ramona
Ladogana, Anna
Puopolo, Maria
Monaco, Salvatore
Agrimi, Umberto
Zanusso, Gianluigi
Pocchiari, Maurizio
JOURNAL OF VIROLOGY, 2017, 91 (11)
[34] The kuru infectious agent is a unique geographic isolate distinct from Creutzfeldt-Jakob disease and scrapie agents
Manuelidis, Laura
Chakrabarty, Trisha
Miyazawa, Kohtaro
Nduom, Nana-Aba
Emmerling, Kaitlin
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2009, 106 (32) : 13529 - 13534
[35] Amyloid Formation: Age-Related Mechanism in Creutzfeldt-Jakob Disease?
Luers, L.
Panza, G.
Henke, F.
Agyenim, T.
Weiss, J.
Willbold, D.
Birkmann, E.
REJUVENATION RESEARCH, 2010, 13 (2-3) : 214 - 216
[36] Different CSF β-amyloid processing in Alzheimer's and Creutzfeldt-Jakob disease
Mollenhauer, Brit
Esselmann, Hermann
Roeber, Sigrun
Schulz-Schaeffer, Walter J.
Trenkwalder, Claudia
Bibl, Mirko
Steinacker, Petra
Kretzschmar, Hans A.
Wiltfang, Jens
Otto, Markus
JOURNAL OF NEURAL TRANSMISSION, 2011, 118 (05) : 691 - 697
[37] Proteomic analysis of host brain components that bind to infectious particles in Creutzfeldt-Jakob disease
Kipkorir, Terry
Colangelo, Christopher M.
Manuelidis, Laura
PROTEOMICS, 2015, 15 (17) : 2983 - 2998
[38] "Preclinical" MSA in definite Creutzfeldt-Jakob disease
Rodriguez-Diehl, Roberta
Jesus Rey, Maria
Gironell, Alexandre
Martinez-Saez, Elena
Ferrer, Isidre
Sanchez-Valle, Raquel
Jaguee, Jordi
Nos, Carlos
Gelpi, Ellen
NEUROPATHOLOGY, 2012, 32 (02) : 158 - 163
[39] Clusterin solubility and aggregation in Creutzfeldt-Jakob disease
Freixes, M
Puig, B
Rodríguez, A
Torrejón-Escribano, B
Blanco, R
Ferrer, I
ACTA NEUROPATHOLOGICA, 2004, 108 (04) : 295 - 301
[40] Molecular classification of sporadic Creutzfeldt-Jakob disease
Hill, AF
Joiner, S
Wadsworth, JDF
Sidle, KCL
Bell, JE
Budka, H
Ironside, JW
Collinge, J
BRAIN, 2003, 126 : 1333 - 1346

← 1 2 3 4 5 →