NITRIC-OXIDE SYNTHASE AND ANTIMICROBIAL ARMATURE OF HUMAN MACROPHAGES

被引：5

作者：

EIZIRIK, DL

机构：

[1] Department of Medical Cell Biology, Uppsala University, Uppsala

来源：

JOURNAL OF INFECTIOUS DISEASES | 1994年 / 170卷 / 03期

关键词：

D O I：

10.1093/infdis/170.3.744

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

[No abstract available]

引用

页码：744 / 745

页数：2

共 10 条

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[2] INTERLEUKIN-1-BETA INDUCES THE FORMATION OF NITRIC-OXIDE BY BETA-CELLS PURIFIED FROM RODENT ISLETS OF LANGERHANS - EVIDENCE FOR THE BETA-CELL AS A SOURCE AND SITE OF ACTION OF NITRIC-OXIDE [J].

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[3] INTERLEUKIN-1-BETA INDUCES THE EXPRESSION OF AN ISOFORM OF NITRIC-OXIDE SYNTHASE IN INSULIN-PRODUCING CELLS, WHICH IS SIMILAR TO THAT OBSERVED IN ACTIVATED MACROPHAGES [J].

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[4] PREDOMINANCE OF STIMULATORY EFFECTS OF INTERLEUKIN-1-BETA ON ISOLATED HUMAN PANCREATIC-ISLETS [J].

EIZIRIK, DL ;

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[5] CYTOKINES SUPPRESS HUMAN ISLET FUNCTION IRRESPECTIVE OF THEIR EFFECTS ON NITRIC-OXIDE GENERATION [J].

EIZIRIK, DL ;

SANDLER, S ;

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[6] INTERLEUKIN-1-INDUCED EXPRESSION OF NITRIC-OXIDE SYNTHASE IN INSULIN-PRODUCING CELLS IS PRECEDED BY C-FOS INDUCTION AND DEPENDS ON GENE-TRANSCRIPTION AND PROTEIN-SYNTHESIS [J].

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[7]

FUCHS D, 1994, J INFECT DIS, V169, P224, DOI 10.1093/infdis/169.1.224

[8] NITRIC-OXIDE SYNTHASE IS NOT A CONSTITUENT OF THE ANTIMICROBIAL ARMATURE OF HUMAN MONONUCLEAR PHAGOCYTES [J].

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[9]

SCHNEEMANN M, 1994, J INFECT DIS, P224

[10] INHIBITION OF INSULIN-SECRETION BY INTERLEUKIN-1-BETA AND TUMOR-NECROSIS-FACTOR-ALPHA VIA AN L-ARGININE-DEPENDENT NITRIC-OXIDE GENERATING MECHANISM [J].

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