EFFECT OF CONVERTING ENZYME-INHIBITION ON GLUCOCORTICOID HYPERTENSION IN THE RAT

被引：42
作者：
ELIJOVICH, F
KRAKOFF, LR
机构：
来源：
AMERICAN JOURNAL OF PHYSIOLOGY | 1980年 / 238卷 / 06期
关键词：
D O I：
10.1152/ajpheart.1980.238.6.H844
中图分类号：
Q4 [生理学];
学科分类号：
071003 ;
摘要：
引用
收藏
页码：H844 / H848
页数：5
相关论文
共 16 条
[1]  
AGREST A, 1974, MEDICINA-BUENOS AIRE, V34, P457
[2]   EVIDENCE FOR AN ANGIOTENSINOGENIC MECHANISM OF HYPERTENSION OF CUSHINGS-SYNDROME [J].
DALAKOS, TG ;
ELIAS, AN ;
ANDERSON, GH ;
STREETEN, DHP ;
SCHROEDER, ET .
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1978, 46 (01) :114-118
[3]   EFFECT OF METHYLPREDNISOLONE ON RENAL FUNCTION AND ZONAL DISTRIBUTION OF BLOOD-FLOW IN RAT [J].
DEBERMUDEZ, L ;
HAYSLETT, JP .
CIRCULATION RESEARCH, 1972, 31 (01) :44-+
[4]  
DREW GM, 1971, ARCH INT PHARMACOD T, V191, P255
[5]   COMPARATIVE-STUDY ON DEVELOPMENT OF CORTICOSTERONE AND DOCA HYPERTENSION IN RATS [J].
HAACK, D ;
MOHRING, J ;
MOHRING, B ;
PETRI, M ;
HACKENTHAL, E .
AMERICAN JOURNAL OF PHYSIOLOGY, 1977, 233 (05) :F403-F411
[6]   BLOCKADE AND STIMULATION OF RENAL, ADRENAL, AND VASCULAR ANGIOTENSIN-2 RECEPTORS WITH 1-SAR, 8-ALA ANGIOTENSIN-2 IN NORMAL MAN [J].
HOLLENBERG, NK ;
WILLIAMS, GH ;
BURGER, B ;
ISHIKAWA, I ;
ADAMS, DF .
JOURNAL OF CLINICAL INVESTIGATION, 1976, 57 (01) :39-46
[7]   MECHANISM OF BLOOD-PRESSURE REDUCTION BY TEPROTIDE (SQ-20881) IN RATS [J].
JAEGER, P ;
FERGUSON, RK ;
BRUNNER, HR ;
KIRCHERTZ, EJ ;
GAVRAS, H .
KIDNEY INTERNATIONAL, 1978, 13 (04) :289-296
[8]   MECHANISM OF HYDROCORTISONE POTENTIATION OF RESPONSES TO EPINEPHRINE AND NOREPINEPHRINE IN RABBIT AORTA [J].
KALSNER, S .
CIRCULATION RESEARCH, 1969, 24 (03) :383-&
[9]   DEPLETION OF CARCASS POTASSIUM IN RATS MADE HYPERTENSIVE WITH DESOXYCORTICOSTERONE ACETATE (DCA) AND WITH CORTISONE [J].
KNOWLTON, AI ;
LOEB, EN .
JOURNAL OF CLINICAL INVESTIGATION, 1957, 36 (09) :1295-1300
[10]   EFFECT OF METHYLPREDNISOLONE UPON ARTERIAL-PRESSURE AND RENIN-ANGIOTENSIN SYSTEM IN RAT [J].
KRAKOFF, LR ;
SELVADURAI, R ;
SUTTER, E .
AMERICAN JOURNAL OF PHYSIOLOGY, 1975, 228 (02) :613-617
12