MELATONIN-INDUCED ANALGESIA REVERSED BY NALOXONE

被引：0
作者：
WINTERS, WD [1 ]
LAKIN, ML [1 ]
机构：
[1] UNIV CALIF DAVIS,DEPT PHARMACOL,DAVIS,CA 95616
来源：
FEDERATION PROCEEDINGS | 1980年 / 39卷 / 03期
关键词：
D O I：
暂无
中图分类号：
Q [生物科学];
学科分类号：
07 ; 0710 ; 09 ;
摘要：
引用
收藏
页码：595 / 595
页数：1
相关论文
共 50 条
[21]   MELATONIN-INDUCED ENHANCEMENT OF ANTIBODY-DEPENDENT CELLULAR CYTOTOXICITY [J].
GIORDANO, M ;
PALERMO, MS .
JOURNAL OF PINEAL RESEARCH, 1991, 10 (03) :117-121
[22]   Melatonin-Induced Nocturnal Vasodilatation Contributes to Skin Regeneration Reply [J].
Chang, Yung-Sen ;
Chiang, Bor-Luen .
JAMA PEDIATRICS, 2016, 170 (06) :622-622
[23]   Selective MT2 melatonin receptor antagonist blocks melatonin-induced antinociception in rats [J].
Yu, CX ;
Zhu, CB ;
Xu, SF ;
Cao, XD ;
Wu, GC .
NEUROSCIENCE LETTERS, 2000, 282 (03) :161-164
[24]   Melatonin-Induced Water Stress Tolerance in Plants: Recent Advances [J].
Moustafa-Farag, Mohamed ;
Mahmoud, Ahmed ;
Arnao, Marino B. ;
Sheteiwy, Mohamed S. ;
Dafea, Mohamed ;
Soltan, Mahmoud ;
Elkelish, Amr ;
Hasanuzzaman, Mirza ;
Ai, Shaoying .
ANTIOXIDANTS, 2020, 9 (09) :1-23
[25]   Inhibition of autophagy triggers melatonin-induced apoptosis in glioblastoma cells [J].
Zhou, Nan ;
Wei, Zi Xuan ;
Qi, Zeng Xin .
BMC NEUROSCIENCE, 2019, 20 (01)
[26]   Inhibition of autophagy triggers melatonin-induced apoptosis in glioblastoma cells [J].
Nan Zhou ;
Zi Xuan Wei ;
Zeng Xin Qi .
BMC Neuroscience, 20
[27]   Pharmacologic studies on the mechanism of melatonin-induced vasorelaxation in rat aorta [J].
Weekley, LB .
JOURNAL OF PINEAL RESEARCH, 1995, 19 (03) :133-138
[28]   NALOXONE-INDUCED ANALGESIA IN DIABETIC MICE [J].
KAMEI, J ;
KAWASHIMA, N ;
KASUYA, Y .
EUROPEAN JOURNAL OF PHARMACOLOGY, 1992, 210 (03) :339-341
[29]   REVERSAL OF HYPNOSIS-INDUCED ANALGESIA BY NALOXONE [J].
STEPHENSON, JBP .
LANCET, 1978, 2 (8097) :991-992
[30]   MELATONIN AND RHYTHMIC PHOTORECEPTOR METABOLISM - MELATONIN-INDUCED CONE ELONGATION IS BLOCKED AT HIGH LIGHT-INTENSITY [J].
PIERCE, ME ;
BESHARSE, JC .
BRAIN RESEARCH, 1987, 405 (02) :400-404
12345