THE COMPLEMENT-SYSTEM IN MYOCARDIAL ISCHAEMIA/REPERFUSION INJURY

被引:112
作者
KILGORE, KS [1 ]
FRIEDRICHS, GS [1 ]
HOMEISTER, JW [1 ]
LUCCHESI, BR [1 ]
机构
[1] UNIV MICHIGAN,SCH MED,DEPT PHARMACOL,ANN ARBOR,MI 48109
关键词
COMPLEMENT; MYOCARDIAL INJURY; NEUTROPHILS; COMPLEMENT PROTECTIVE PROTEINS; REPERFUSION;
D O I
10.1093/cvr/28.4.437
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
[No abstract available]
引用
收藏
页码:437 / 444
页数:8
相关论文
共 96 条
[1]   CONTRACTILE ACTIONS OF C5A ON ISOLATED PORCINE MYOCARDIUM [J].
AMSTERDAM, EA ;
RENDIG, SV ;
LONGHURST, JC .
AMERICAN JOURNAL OF PHYSIOLOGY, 1992, 263 (03) :H740-H745
[2]   PHARMACOLOGICAL STUDIES OF FUT-175, NAFAMSTAT MESILATE .1. INHIBITION OF PROTEASE ACTIVITY IN INVITRO AND INVIVO EXPERIMENTS [J].
AOYAMA, T ;
INO, Y ;
OZEKI, M ;
ODA, M ;
SATO, T ;
KOSHIYAMA, Y ;
SUZUKI, S ;
FUJITA, M .
JAPANESE JOURNAL OF PHARMACOLOGY, 1984, 35 (03) :203-227
[3]  
ATKINSON JP, 1991, IMMUNOLOGY, P1111
[4]  
AYESH SK, 1990, J IMMUNOL, V144, P3066
[5]   MYOCARDIAL CONSEQUENCES OF REPERFUSION [J].
BECKER, LC ;
AMBROSIO, G .
PROGRESS IN CARDIOVASCULAR DISEASES, 1987, 30 (01) :23-44
[6]  
BEYERSDORF F, 1989, ARZNEIMITTEL-FORSCH, V39-1, P46
[7]  
BIESECKER G, 1990, J IMMUNOL, V145, P209
[8]   MARKED REDUCTION OF FREE-RADICAL GENERATION AND CONTRACTILE DYSFUNCTION BY ANTIOXIDANT THERAPY BEGUN AT THE TIME OF REPERFUSION - EVIDENCE THAT MYOCARDIAL STUNNING IS A MANIFESTATION OF REPERFUSION INJURY [J].
BOLLI, R ;
JEROUDI, MO ;
PATEL, BS ;
ARUOMA, OI ;
HALLIWELL, B ;
LAI, EK ;
MCCAY, PB .
CIRCULATION RESEARCH, 1989, 65 (03) :607-622
[9]   INFLAMMATORY MEDIATORS RELEASED BY COMPLEMENT-DERIVED PEPTIDES [J].
BULT, H ;
HERMAN, AG .
AGENTS AND ACTIONS, 1983, 13 (5-6) :405-414
[10]   MONOCLONAL-ANTIBODIES DEMONSTRATE PROTECTION OF POLYMORPHONUCLEAR LEUKOCYTES AGAINST COMPLEMENT ATTACK [J].
CAMPBELL, AK ;
MORGAN, BP .
NATURE, 1985, 317 (6033) :164-166