PARENTAL IMPRINTING OF THE H19 AND IGF2 GENES IN THE MOUSE

被引：20

作者：

TILGHMAN, SM ^{[1
]}

BARTOLOMEI, MS ^{[1
]}

WEBBER, AL ^{[1
]}

BRUNKOW, ME ^{[1
]}

SAAM, J ^{[1
]}

LEIGHTON, PA ^{[1
]}

PFEIFER, K ^{[1
]}

ZEMEL, S ^{[1
]}

机构：

[1] PRINCETON UNIV, DEPT MOLEC BIOL, PRINCETON, NJ 08544 USA

来源：

COLD SPRING HARBOR SYMPOSIA ON QUANTITATIVE BIOLOGY | 1993年 / 58卷

关键词：

D O I：

10.1101/SQB.1993.058.01.034

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：287 / 295

页数：9

共 60 条

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[13] THE PRODUCT OF THE MOUSE XIST GENE IS A 15 KB INACTIVE X-SPECIFIC TRANSCRIPT CONTAINING NO CONSERVED ORF AND LOCATED IN THE NUCLEUS [J].

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[15] THE HUMAN XIST GENE - ANALYSIS OF A 17 KB INACTIVE X-SPECIFIC RNA THAT CONTAINS CONSERVED REPEATS AND IS HIGHLY LOCALIZED WITHIN THE NUCLEUS [J].

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[16] ECTOPIC EXPRESSION OF THE H19 GENE IN MICE CAUSES PRENATAL LETHALITY [J].

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[17] DNA METHYLATION AND THE REGULATION OF GLOBIN GENE-EXPRESSION [J].

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[18] A CANDIDATE MOUSE MODEL FOR PRADER-WILLI SYNDROME WHICH SHOWS AN ABSENCE OF SNRPN EXPRESSION [J].

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[19] PARENTAL-SPECIFIC METHYLATION OF AN IMPRINTED TRANSGENE IS ESTABLISHED DURING GAMETOGENESIS AND PROGRESSIVELY CHANGES DURING EMBRYOGENESIS [J].

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[20] A GROWTH-DEFICIENCY PHENOTYPE IN HETEROZYGOUS MICE CARRYING AN INSULIN-LIKE GROWTH FACTOR-II GENE DISRUPTED BY TARGETING [J].

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ROBERTSON, EJ .

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